(Q28592886)

English

Potassium restriction, high protein intake, and metabolic acidosis increase expression of the glutamine transporter SNAT3 (Slc38a3) in mouse kidney

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scholarly article

1 reference

PubMed publication ID

31 January 2017

Potassium restriction, high protein intake, and metabolic acidosis increase expression of the glutamine transporter SNAT3 (Slc38a3) in mouse kidney (English)

1 reference

PubMed publication ID

31 January 2017

Phosphoenolpyruvate carboxykinase 1, cytosolic

1 reference

GOA release 2020-03-11

Solute carrier family 38, member 3

1 reference

GOA release 2020-03-11

metabolic acidosis

1 reference

based on heuristic

inferred from title

author name string

Stephanie M. Busque

1

1 reference

PubMed publication ID

31 January 2017

Carsten A. Wagner

2

1 reference

PubMed publication ID

31 January 2017

publication date

1 August 2009

1 reference

PubMed publication ID

31 January 2017

American Journal of Physiology - Renal Physiology

1 reference

PubMed publication ID

31 January 2017

297

1 reference

PubMed publication ID

31 January 2017

2

1 reference

PubMed publication ID

31 January 2017

F440–450

1 reference

PubMed publication ID

31 January 2017

Renal metabolic response to acid base changes. I. Enzymatic control of ammoniagenesis in the rat.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Functional cooperation of epithelial heteromeric amino acid transporters expressed in madin-darby canine kidney cells.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Regulation of the glutamine transporter SN1 by extracellular pH and intracellular sodium ions

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Molecular analysis of system N suggests novel physiological roles in nitrogen metabolism and synaptic transmission

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Phosphate-dependent glutaminase from rat kidney. Cause of increased activity in response to acidosis and identity with glutaminase from other tissues

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Regulation of expression of the SN1 transporter during renal adaptation to chronic metabolic acidosis in rats

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Sodium-coupled neutral amino acid (System N/A) transporters of the SLC38 gene family

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Regulation of renal amino acid transporters during metabolic acidosis.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Effect of bath and luminal potassium concentration on ammonia production and secretion by mouse proximal tubules perfused in vitro

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Intra- and inter-nephron heterogeneity of ammoniagenesis in rats: effects of chronic metabolic acidosis and potassium depletion.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Relation of renal cortical gluconeogenesis, glutamate content, and production of ammonia

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Glucose and glutamine utilization by rat lymphocytes, monocytes and neutrophils in culture: a comparative study.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Induction and targeting of the glutamine transporter SN1 to the basolateral membranes of cortical kidney tubule cells during chronic metabolic acidosis suggest a role in pH regulation

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Potassium depletion increases luminal Na+/H+ exchange and basolateral Na+:CO3=:HCO3- cotransport in rat renal cortex

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Localization and regulation of the ATP6V0A4 (a4) vacuolar H+-ATPase subunit defective in an inherited form of distal renal tubular acidosis

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

The evolutionary history and tissue mapping of amino acid transporters belonging to solute carrier families SLC32, SLC36, and SLC38.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

The effect of uncomplicated potassium depletion on urine acidification

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Relationship of renal ammonia production and potassium homeostasis

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Regulation of the expression of the Cl-/anion exchanger pendrin in mouse kidney by acid-base status

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Mechanisms of NH4+ and NH3 transport during hypokalemia

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Endogenous endothelins mediate increased distal tubule acidification induced by dietary acid in rats

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Increased Endothelin Activity Mediates Augmented Distal Nephron Acidification Induced by Dietary Protein

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Dietary protein induces endothelin-mediated kidney injury through enhanced intrinsic acid production

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.90318.2008

21 January 2018

Identifiers

10.1152/AJPRENAL.90318.2008

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

PubMed publication ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

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