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DHFR/MSH3 amplification in methotrexate-resistant cells alters the hMutSalpha/hMutSbeta ratio and reduces the efficiency of base-base mismatch repair
scientific journal article
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scholarly article
1 reference
stated in
PubMed
PubMed ID
9294177
retrieved
3 February 2017
title
DHFR/MSH3 amplification in methotrexate-resistant cells alters the hMutSalpha/hMutSbeta ratio and reduces the efficiency of base-base mismatch repair
(English)
1 reference
stated in
PubMed
PubMed ID
9294177
retrieved
3 February 2017
main subject
SH3 domain
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methotrexate
1 reference
based on heuristic
inferred from title
DNA mismatch repair
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based on heuristic
inferred from title
author name string
J. T. Drummond
series ordinal
1
1 reference
stated in
PubMed
PubMed ID
9294177
retrieved
3 February 2017
J. Genschel
series ordinal
2
1 reference
stated in
PubMed
PubMed ID
9294177
retrieved
3 February 2017
E. Wolf
series ordinal
3
1 reference
stated in
PubMed
PubMed ID
9294177
retrieved
3 February 2017
P. Modrich
series ordinal
4
1 reference
stated in
PubMed
PubMed ID
9294177
retrieved
3 February 2017
language of work or name
English
0 references
publication date
16 September 1997
1 reference
stated in
PubMed
PubMed ID
9294177
retrieved
3 February 2017
published in
Proceedings of the National Academy of Sciences of the United States of America
1 reference
stated in
PubMed
PubMed ID
9294177
retrieved
3 February 2017
volume
94
1 reference
stated in
PubMed
PubMed ID
9294177
retrieved
3 February 2017
page(s)
10144–10149
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stated in
PubMed
PubMed ID
9294177
retrieved
3 February 2017
issue
19
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PubMed
PubMed ID
9294177
retrieved
3 February 2017
cites work
Mutations of Bacteria from Virus Sensitivity to Virus Resistance
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PubMed Central
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hMSH2 forms specific mispair-binding complexes with hMSH3 and hMSH6
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PubMed Central
reference URL
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Binding of insertion/deletion DNA mismatches by the heterodimer of yeast mismatch repair proteins MSH2 and MSH3.
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17 March 2017
hMutSbeta, a heterodimer of hMSH2 and hMSH3, binds to insertion/deletion loops in DNA
1 reference
stated in
PubMed Central
reference URL
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Mutation of MSH3 in endometrial cancer and evidence for its functional role in heteroduplex repair
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The yeast gene MSH3 defines a new class of eukaryotic MutS homologues.
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Human strand-specific mismatch repair occurs by a bidirectional mechanism similar to that of the bacterial reaction
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Identification of two initiator elements in the bidirectional promoter of the human dihydrofolate reductase and mismatch repair protein 1 genes
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Isolation of an hMSH2-p160 heterodimer that restores DNA mismatch repair to tumor cells
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Isolation and characterization of cDNA clones derived from the divergently transcribed gene in the region upstream from the human dihydrofolate reductase gene
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Redundancy of Saccharomyces cerevisiae MSH3 and MSH6 in MSH2-dependent mismatch repair
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Crossref
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Folate deficiency causes uracil misincorporation into human DNA and chromosome breakage: implications for cancer and neuronal damage
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Human MutSalpha recognizes damaged DNA base pairs containing O6-methylguanine, O4-methylthymine, or the cisplatin-d(GpG) adduct
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An alkylation-tolerant, mutator human cell line is deficient in strand-specific mismatch repair.
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Mutator phenotypes in human colorectal carcinoma cell lines
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Loss of expression of the human MSH3 gene in hematological malignancies
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Mutations in the MSH3 gene preferentially lead to deletions within tracts of simple repetitive DNA in Saccharomyces cerevisiae
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PubMed Central
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Cytologic evidence for gene amplification in methotrexate-resistant cells obtained from a patient with ovarian adenocarcinoma
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Gene amplification in a leukemic patient treated with methotrexate
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Unstable Methotrexate Resistance in Human Small-Cell Carcinoma Associated with Double Minute Chromosomes
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Frameshift mutations and the genetic code. This paper is dedicated to Professor Theodosius Dobzhansky on the occasion of his 66th birthday
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Dual bidirectional promoters at the mouse dhfr locus: cloning and characterization of two mRNA classes of the divergently transcribed Rep-1 gene
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Increased rate of base substitution in a hamster mutator strain obtained during serial selection for gene amplification
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Hypermutability and mismatch repair deficiency in RER+ tumor cells
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Defective mismatch repair in extracts of colorectal and endometrial cancer cell lines exhibiting microsatellite instability
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3 June 2018
A mutator phenotype characterizes one of two complementation groups in human cells tolerant to methylation damage.
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PubMed Central
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3 June 2018
The bidirectionally transcribed dihydrofolate reductase and rep-3a promoters are growth regulated by distinct mechanisms.
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PubMed Central
reference URL
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3 June 2018
A table for the estimation of the spontaneous mutations rate of cells in culture
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3 June 2018
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3 June 2018
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3 June 2018
Requirement of the yeast MSH3 and MSH6 genes for MSH2-dependent genomic stability
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/9294177
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Inhibition of dihydrofolic reductase by aminopterin and amethopterin
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/9294177
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1073/PNAS.94.19.10144
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4519055
ADS bibcode
1997PNAS...9410144D
0 references
OpenCitations bibliographic resource ID
4519055
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4519055
PMCID
23329
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4519055
PubMed ID
9294177
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4519055
ResearchGate publication ID
13927385
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