(Q29300293)

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Pro-inflammatory programmed cell death

scientific article (publication date: March 2001)

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Pro-inflammatory programmed cell death (English)

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https://doi.org/10.1016%2FS0966-842X%2800%2901936-3

9 April 2017

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Brad T Cookson

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Molly A Brennan

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March 2001

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Trends in Microbiology

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9

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3

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113-114

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Salmonella induces macrophage death by caspase-1-dependent necrosis.

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The Salmonella invasin SipB induces macrophage apoptosis by binding to caspase-1

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Salmonella-induced cell death: apoptosis, necrosis or programmed cell death?

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Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

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Activation of an interleukin 1 converting enzyme-dependent apoptosis pathway by granzyme B.

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Expression of a dominant negative mutant of interleukin-1 beta converting enzyme in transgenic mice prevents neuronal cell death induced by trophic factor withdrawal and ischemic brain injury

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Attenuation of transient focal cerebral ischemic injury in transgenic mice expressing a mutant ICE inhibitory protein

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Mice deficient in interleukin-1 converting enzyme are resistant to neonatal hypoxic-ischemic brain damage

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Localization of apoptotic macrophages at the site of plaque rupture in sudden coronary death

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Salmonella exploits caspase-1 to colonize Peyer's patches in a murine typhoid model

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(Q29300293)

Pro-inflammatory programmed cell death

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