(Q31110982)

English

Truncated KCNQ1 mutant, A178fs/105, forms hetero-multimer channel with wild-type causing a dominant-negative suppression due to trafficking defect

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Europe PubMed Central

28 June 2017

Truncated KCNQ1 mutant, A178fs/105, forms hetero-multimer channel with wild-type causing a dominant-negative suppression due to trafficking defect (English)

1 reference

Europe PubMed Central

28 June 2017

author name string

Yoshiyasu Aizawa

1

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Europe PubMed Central

28 June 2017

Kazuo Ueda

2

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Europe PubMed Central

28 June 2017

Long-Mei Wu

3

1 reference

Europe PubMed Central

28 June 2017

Natsuko Inagaki

4

1 reference

Europe PubMed Central

28 June 2017

Takeharu Hayashi

5

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Europe PubMed Central

28 June 2017

Megumi Takahashi

6

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Europe PubMed Central

28 June 2017

Masaaki Ohta

7

1 reference

Europe PubMed Central

28 June 2017

Seiko Kawano

8

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Europe PubMed Central

28 June 2017

Yuji Hirano

9

1 reference

Europe PubMed Central

28 June 2017

Michio Yasunami

10

1 reference

Europe PubMed Central

28 June 2017

Yoshifusa Aizawa

11

1 reference

Europe PubMed Central

28 June 2017

Akinori Kimura

12

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Europe PubMed Central

28 June 2017

Masayasu Hiraoka

13

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28 June 2017

language of work or name

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publication date

1 September 2004

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Europe PubMed Central

28 June 2017

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Europe PubMed Central

28 June 2017

574

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28 June 2017

1-3

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145-150

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Site-directed mutagenesis by overlap extension using the polymerase chain reaction

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.08.018

21 January 2018

Effects of Na+ channel blocker, pilsicainide, on HERG current expressed in HEK-293 cells

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.08.018

21 January 2018

Defective protein trafficking in hERG-associated hereditary long QT syndrome (LQT2): molecular mechanisms and restoration of intracellular protein processing

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.08.018

21 January 2018

Interaction with GM130 during HERG ion channel trafficking. Disruption by type 2 congenital long QT syndrome mutations. Human Ether-à-go-go-Related Gene.

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.08.018

21 January 2018

Characterization and subcellular localization of KCNQ1 with a heterozygous mutation in the C terminus

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.08.018

21 January 2018

Molecular biology of arrhythmic syndromes

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.08.018

21 January 2018

Pathophysiological mechanisms of dominant and recessive KVLQT1 K+ channel mutations found in inherited cardiac arrhythmias.

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.08.018

21 January 2018

Retention in the endoplasmic reticulum as a mechanism of dominant-negative current suppression in human long QT syndrome

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.08.018

21 January 2018

A recessive C-terminal Jervell and Lange-Nielsen mutation of the KCNQ1 channel impairs subunit assembly

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.08.018

21 January 2018

A carboxy-terminal domain determines the subunit specificity of KCNQ K+ channel assembly

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2004.08.018

21 January 2018

Identifiers

10.1016/J.FEBSLET.2004.08.018

1 reference

Europe PubMed Central

28 June 2017

1 reference

Europe PubMed Central

28 June 2017

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