(Q31722034)

3 July 2017

title

Increased inactivation of nitric oxide is involved in coronary endothelial dysfunction in heart failure (English)

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3 July 2017

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Arimura K

1

1 reference

3 July 2017

Egashira K

2

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3 July 2017

Nakamura R

3

1 reference

3 July 2017

Ide T

4

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3 July 2017

Tsutsui H

5

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3 July 2017

Shimokawa H

6

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3 July 2017

Takeshita A

7

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3 July 2017

publication date

1 January 2001

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American Journal of Physiology Heart and Circulatory Physiology

3 July 2017

published in

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3 July 2017

volume

280

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3 July 2017

issue

1

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3 July 2017

page(s)

H68-75

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Oxygen free radicals and congestive heart failure

3 July 2017

cites work

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

Endothelial dysfunction: clinical implications

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

Superoxide anion radical (O2-.), superoxide dismutases, and related matters

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

Chronic heart failure in the United States: a manifestation of coronary artery disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

Cellular and molecular mechanisms of endothelial cell dysfunction

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

LDLs impair vasomotor function of the coronary microcirculation: role of superoxide anions

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

Effects of L-arginine on impaired acetylcholine-induced and ischemic vasodilation of the forearm in patients with heart failure.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

The role of endothelium-derived vasoactive substances in the pathophysiology of exercise intolerance in patients with congestive heart failure.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

The catecholic metal sequestering agent 1,2-dihydroxybenzene-3, 5-disulfonate confers protection against oxidative cell damage

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

Pacing-induced heart failure in dogs enhances the expression of p53 and p53-dependent genes in ventricular myocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

Angiotensin II in the evolution of experimental heart failure.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

Evidence for enhanced vascular superoxide anion production in nitrate tolerance. A novel mechanism underlying tolerance and cross-tolerance

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

Hypercholesterolemia increases endothelial superoxide anion production

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

Endothelium-dependent dilation of the coronary microvasculature is impaired in dilated cardiomyopathy.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

Role of myocyte nitric oxide in beta-adrenergic hyporesponsiveness in heart failure

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

Immunohistochemical detection of 4-hydroxynonenal protein adducts in Parkinson disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

Elevated Levels of 8-iso-Prostaglandin F 2α in Pericardial Fluid of Patients With Heart Failure

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.1.H68

10.1152/AJPHEART.2001.280.1.H68

21 January 2018

Identifiers

DOI

0 references

1 reference