(Q32063589)

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N-methyl-D-aspartate receptor-mediated mitochondrial Ca(2+) overload in acute excitotoxic motor neuron death: a mechanism distinct from chronic neurotoxicity after Ca(2+) influx

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

N-methyl-D-aspartate receptor-mediated mitochondrial Ca(2+) overload in acute excitotoxic motor neuron death: a mechanism distinct from chronic neurotoxicity after Ca(2+) influx (English)

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

1 reference

based on heuristic

inferred from title

1 reference

based on heuristic

inferred from title

toxic encephalopathy

1 reference

based on heuristic

inferred from title

author name string

M Urushitani

1

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

T Nakamizo

2

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

R Inoue

3

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

H Sawada

4

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

T Kihara

5

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

K Honda

6

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

A Akaike

7

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

S Shimohama

8

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

publication date

1 March 2001

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

Journal of Neuroscience Research

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

63

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

5

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

377-387

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

Calcineurin and mitochondrial function in glutamate-induced neuronal cell death.

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Calcium enhances in vitro free radical-induced damage to brain synaptosomes, mitochondria, and cultured spinal cord neurons

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Recruitment of mitochondrial cyclophilin to the mitochondrial inner membrane under conditions of oxidative stress that enhance the opening of a calcium-sensitive non-specific channel

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Trifluoperazine and dibucaine-induced inhibition of glutamate-induced mitochondrial depolarization in rat cultured forebrain neurones

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Generation of reactive oxygen species, release of L-glutamate and activation of caspases are required for oxygen-induced apoptosis of embryonic hippocampal neurons in culture.

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Regulation of AMPA receptor-mediated synaptic transmission by clathrin-dependent receptor internalization

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Effects of NMDA receptor antagonists following spinal ischemia in the rabbit

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21 January 2018

Mitochondria and neuronal glutamate excitotoxicity

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Visualization of NMDA receptor-induced mitochondrial calcium accumulation in striatal neurons

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2,7-Dihydrodichlorofluorescein diacetate as a fluorescent marker for peroxynitrite formation

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Therapeutic horizons for amyotrophic lateral sclerosis

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Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity

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21 January 2018

NMDA-induced superoxide production and neurotoxicity in cultured rat hippocampal neurons: role of mitochondria

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Calcium-permeable alpha-amino-3-hydroxy-5-methyl-4-isoxazole propionic acid receptors: a molecular determinant of selective vulnerability in amyotrophic lateral sclerosis

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Glutamate or aspartate as a possible neurotransmitter of cerebral corticofugal fibers in the monkey

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In vitro kainate injury to large, SMI-32(+) spinal neurons is Ca2+ dependent.

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Activation of calpain I converts excitotoxic neuron death into a caspase-independent cell death

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Identifiers

10.1002/1097-4547(20010301)63:5<377::AID-JNR1032>3.0.CO;2-#

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

6 July 2017

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