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Deciphering the pathways of life and death
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scholarly article
1 reference
stated in
Europe PubMed Central
PubMed ID
10209152
retrieved
28 July 2017
review article
1 reference
stated in
Europe PubMed Central
title
Deciphering the pathways of life and death
(English)
1 reference
stated in
Europe PubMed Central
PubMed ID
10209152
retrieved
28 July 2017
author name string
Li H
series ordinal
1
1 reference
stated in
Europe PubMed Central
PubMed ID
10209152
retrieved
28 July 2017
Yuan J
series ordinal
2
1 reference
stated in
Europe PubMed Central
PubMed ID
10209152
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28 July 2017
publication date
1 April 1999
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stated in
Europe PubMed Central
PubMed ID
10209152
retrieved
28 July 2017
published in
Current Opinion in Cell Biology
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stated in
Europe PubMed Central
PubMed ID
10209152
retrieved
28 July 2017
volume
11
1 reference
stated in
Europe PubMed Central
PubMed ID
10209152
retrieved
28 July 2017
page(s)
261-266
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stated in
Europe PubMed Central
PubMed ID
10209152
retrieved
28 July 2017
issue
2
1 reference
stated in
Europe PubMed Central
PubMed ID
10209152
retrieved
28 July 2017
cites work
Proteases to die for
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Solution structure of the RAIDD CARD and model for CARD/CARD interaction in caspase-2 and caspase-9 recruitment
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Crossref
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NMR structure and mutagenesis of the FADD (Mort1) death-effector domain
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Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3
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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade
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Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c
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Autoactivation of procaspase-9 by Apaf-1-mediated oligomerization
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Essential role of CED-4 oligomerization in CED-3 activation and apoptosis
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Death receptors: signaling and modulation
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7 January 2021
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An induced proximity model for caspase-8 activation.
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Targeted disruption of the mouse Caspase 8 gene ablates cell death induction by the TNF receptors, Fas/Apo1, and DR3 and is lethal prenatally.
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Fas-mediated apoptosis and activation-induced T-cell proliferation are defective in mice lacking FADD/Mort1
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FADD: essential for embryo development and signaling from some, but not all, inducers of apoptosis
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reference URL
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Reduced apoptosis and cytochrome c-mediated caspase activation in mice lacking caspase 9
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Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice
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7 January 2021
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Essential contribution of caspase 3/CPP32 to apoptosis and its associated nuclear changes
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7 January 2021
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inferred from DOI database lookup
Differential requirement for caspase 9 in apoptotic pathways in vivo.
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reference URL
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7 January 2021
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Apaf1 is required for mitochondrial pathways of apoptosis and brain development
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reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
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Apaf1 (CED-4 homolog) regulates programmed cell death in mammalian development
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7 January 2021
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The C. elegans protein EGL-1 is required for programmed cell death and interacts with the Bcl-2-like protein CED-9
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https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis
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BID: a novel BH3 domain-only death agonist
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https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors
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reference URL
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Molecular cloning of a novel polypeptide, DP5, induced during programmed neuronal death
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inferred from DOI database lookup
Bax involvement in p53-mediated neuronal cell death
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Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)
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Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery
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Interleukin-3-induced phosphorylation of BAD through the protein kinase Akt
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Crossref
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Movement of Bax from the cytosol to mitochondria during apoptosis
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Bcl-2 and Bax function independently to regulate cell death
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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based on heuristic
inferred from DOI database lookup
Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
based on heuristic
inferred from DOI database lookup
Defects in regulation of apoptosis in caspase-2-deficient mice
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
based on heuristic
inferred from DOI database lookup
Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
based on heuristic
inferred from DOI database lookup
Cloning of a bcl-2 homologue by interaction with adenovirus E1B 19K
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
based on heuristic
inferred from DOI database lookup
Induction of apoptosis by the Bcl-2 homologue Bak
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
based on heuristic
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Modulation of apoptosis by the widely distributed Bcl-2 homologue Bak
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
based on heuristic
inferred from DOI database lookup
Bok is a pro-apoptotic Bcl-2 protein with restricted expression in reproductive tissues and heterodimerizes with selective anti-apoptotic Bcl-2 family members
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
based on heuristic
inferred from DOI database lookup
Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
based on heuristic
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Induction of apoptosis by human Nbk/Bik, a BH3-containing protein that interacts with E1B 19K
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
based on heuristic
inferred from DOI database lookup
Blk, a BH3-containing mouse protein that interacts with Bcl-2 and Bcl-xL, is a potent death agonist
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
based on heuristic
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harakiri, a novel regulator of cell death, encodes a protein that activates apoptosis and interacts selectively with survival-promoting proteins Bcl-2 and Bcl-X(L)
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
based on heuristic
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Bim: a novel member of the Bcl-2 family that promotes apoptosis
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
based on heuristic
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BOD (Bcl-2-related ovarian death gene) is an ovarian BH3 domain-containing proapoptotic Bcl-2 protein capable of dimerization with diverse antiapoptotic Bcl-2 members
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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based on heuristic
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The E1B 19K/Bcl-2-binding protein Nip3 is a dimeric mitochondrial protein that activates apoptosis
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2899%2980035-0
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7 January 2021
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Identifiers
DOI
10.1016/S0955-0674(99)80035-0
1 reference
stated in
Europe PubMed Central
PubMed ID
10209152
retrieved
28 July 2017
PubMed ID
10209152
1 reference
stated in
Europe PubMed Central
PubMed ID
10209152
retrieved
28 July 2017
ResearchGate publication ID
13089468
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