(Q33670203)

28 July 2017

1 reference

title

Two MAD tails: what the recent knockouts of Mad1 and Mxi1 tell us about the MYC/MAX/MAD network (English)

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28 July 2017

K P Foley

1

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28 July 2017

R N Eisenman

series ordinal

2

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28 July 2017

language of work or name

English

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publication date

1 May 1999

1 reference

Biochimica et Biophysica Acta

28 July 2017

published in

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28 July 2017

volume

1423

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28 July 2017

issue

3

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28 July 2017

page(s)

M37-47

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The Mad protein family links transcriptional repression to cell differentiation

28 July 2017

cites work

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Mad: a heterodimeric partner for Max that antagonizes Myc transcriptional activity

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Mxi1, a protein that specifically interacts with Max to bind Myc-Max recognition sites

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Max: a helix-loop-helix zipper protein that forms a sequence-specific DNA-binding complex with Myc

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Recognition by Max of its cognate DNA through a dimeric b/HLH/Z domain

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Binding of myc proteins to canonical and noncanonical DNA sequences

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Mnt, a novel Max-interacting protein is coexpressed with Myc in proliferating cells and mediates repression at Myc binding sites

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Myc and Max associate in vivo

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

A switch from Myc:Max to Mad:Max heterocomplexes accompanies monocyte/macrophage differentiation.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Myc-Max-Mad: a transcription factor network controlling cell cycle progression, differentiation and death

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

An amino-terminal c-myc domain required for neoplastic transformation activates transcription

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Myc and Max proteins possess distinct transcriptional activities

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Transcriptional activation by the human c-Myc oncoprotein in yeast requires interaction with Max.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Expression of the mad gene during cell differentiation in vivo and its inhibition of cell growth in vitro

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Expression of Mad, an antagonist of Myc oncoprotein function, in differentiating keratinocytes during tumorigenesis of the skin

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Sequential expression of the MAD family of transcriptional repressors during differentiation and development.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Function of the c-Myc antagonist Mad1 during a molecular switch from proliferation to differentiation

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Targeted disruption of the MYC antagonist MAD1 inhibits cell cycle exit during granulocyte differentiation.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Suppression of Myc, but not E1a, transformation activity by Max-associated proteins, Mad and Mxi1

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Effects of the MYC oncogene antagonist, MAD, on proliferation, cell cycling and the malignant phenotype of human brain tumour cells

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Inhibition of cell proliferation by the Mad1 transcriptional repressor

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Inducible expression of Mad accelerates growth arrest of serum deprived human glioblastoma cells.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Repression of c-Myc responsive genes in cycling cells causes G1 arrest through reduction of cyclin E/CDK2 kinase activity.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Mutation of the MXI1 gene in prostate cancer

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Allelic loss on chromosome 10q in human lung cancer: association with tumour progression and metastatic phenotype

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

PTEN, a putative protein tyrosine phosphatase gene mutated in human brain, breast, and prostate cancer

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Mad-Max transcriptional repression is mediated by ternary complex formation with mammalian homologs of yeast repressor Sin3

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

An amino-terminal domain of Mxi1 mediates anti-Myc oncogenic activity and interacts with a homolog of the yeast transcriptional repressor SIN3

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Mad proteins contain a dominant transcription repression domain.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Role for N-CoR and histone deacetylase in Sin3-mediated transcriptional repression

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Histone deacetylase activity is required for full transcriptional repression by mSin3A

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

A complex containing N-CoR, mSin3 and histone deacetylase mediates transcriptional repression

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Histone deacetylases associated with the mSin3 corepressor mediate mad transcriptional repression

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Nuclear receptor repression mediated by a complex containing SMRT, mSin3A, and histone deacetylase

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Histone deacetylases and SAP18, a novel polypeptide, are components of a human Sin3 complex

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Targeting chromatin disruption: Transcription regulators that acetylate histones

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Histone acetylation in chromatin structure and transcription

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Transcriptional control. Sinful repression

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Methylated DNA and MeCP2 recruit histone deacetylase to repress transcription

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Transcriptional repression by the methyl-CpG-binding protein MeCP2 involves a histone deacetylase complex

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Role of Mxi1 in ageing organ systems and the regulation of normal and neoplastic growth

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Traps to catch unwary oncogenes

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

The molecular control of cell division, differentiation commitment and maturation in haemopoietic cells

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Functional aspects of apoptosis in hematopoiesis and consequences of failure

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Loss of N-myc function results in embryonic lethality and failure of the epithelial component of the embryo to develop

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Translocations among antibody genes in human cancer

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Mxi1 tumor suppressor gene is not mutated in primary pancreatic adenocarcinoma

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Identification of two distinct tumor-suppressor loci on the long arm of chromosome 10 in small cell lung cancer

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

No germline mutations in the dimerization domain of MXI1 in prostate cancer clusters. The CRC/BPG UK Familial Prostate Cancer Study Collaborators. Cancer Research Campaign/British Prostate Group

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

Point mutations of the Mxil gene are rare in prostate cancers

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

c-myc null cells misregulate cad and gadd45 but not other proposed c-Myc targets

1 reference

https://api.crossref.org/works/10.1016%2FS0304-419X%2899%2900012-8

7 January 2021

10.1016/S0304-419X(99)00012-8

Identifiers

DOI

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28 July 2017

1 reference