(Q34052929)

30 July 2017

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title

Mitochondria: execution central (English)

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30 July 2017

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Gottlieb RA

1

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30 July 2017

language of work or name

English

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publication date

1 September 2000

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30 July 2017

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30 July 2017

volume

482

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30 July 2017

issue

1-2

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30 July 2017

page(s)

6-12

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Electron microscopic tomography of rat-liver mitochondria and their interaction with the endoplasmic reticulum

30 July 2017

cites work

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Electron Tomography of Neuronal Mitochondria: Three-Dimensional Structure and Organization of Cristae and Membrane Contacts

7 January 2021

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Mitochondria and cell death. Mechanistic aspects and methodological issues

7 January 2021

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Chloromethyltetramethylrosamine (Mitotracker Orange) induces the mitochondrial permeability transition and inhibits respiratory complex I. Implications for the mechanism of cytochrome c release.

7 January 2021

1 reference

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The caspase-3 precursor has a cytosolic and mitochondrial distribution: implications for apoptotic signaling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Mitochondrial release of caspase-2 and -9 during the apoptotic process

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Electrophysiological study of a novel large pore formed by Bax and the voltage-dependent anion channel that is permeable to cytochrome c.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Outer mitochondrial membrane permeability can regulate coupled respiration and cell survival

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Protein transport into mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Evidence for the Involvement of a Membrane-Associated Cyclosporin-A-Binding Protein in the Ca2+-Activated Inner Membrane Pore of Heart Mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Bcl-2 family: life-or-death switch

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Protein phosphatase 1alpha is a Ras-activated Bad phosphatase that regulates interleukin-2 deprivation-induced apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Ca2+-induced apoptosis through calcineurin dephosphorylation of BAD

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Calphostin C-mediated translocation and integration of Bax into mitochondria induces cytochrome c release before mitochondrial dysfunction.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Integrin-mediated survival signals regulate the apoptotic function of Bax through its conformation and subcellular localization.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

X-ray and NMR structure of human Bcl-xL, an inhibitor of programmed cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Bcl-2 family proteins as ion-channels

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Comparison of the ion channel characteristics of proapoptotic BAX and antiapoptotic BCL-2

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Bcl-xL prevents cell death following growth factor withdrawal by facilitating mitochondrial ATP/ADP exchange

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Regulation of the permeability transition pore in skeletal muscle mitochondria. Modulation By electron flow through the respiratory chain complex i.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Cyclophilin-D binds strongly to complexes of the voltage-dependent anion channel and the adenine nucleotide translocase to form the permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Bcl-2 and Bax regulate the channel activity of the mitochondrial adenine nucleotide translocator

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Collapse of the inner mitochondrial transmembrane potential is not required for apoptosis of HL60 cells.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Release of mitochondrial cytochrome c is upstream of caspase activation in chemical-induced apoptosis in human monocytic tumour cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Loss of function of cytochrome c in Jurkat cells undergoing fas-mediated apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Lack of release of cytochrome C from mitochondria into cytosol early in the course of Fas-mediated apoptosis of Jurkat cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Bcl-2 and the Outer Mitochondrial Membrane in the Inactivation of Cytochrome c during Fas-mediated Apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Bax directly induces release of cytochrome c from isolated mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

The pro-apoptotic proteins, Bid and Bax, cause a limited permeabilization of the mitochondrial outer membrane that is enhanced by cytosol

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

An induced proximity model for caspase-8 activation.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

DFF, a heterodimeric protein that functions downstream of caspase-3 to trigger DNA fragmentation during apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Activation of Fas by FasL induces apoptosis by a mechanism that cannot be blocked by Bcl-2 or Bcl-x(L)

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Mechanisms of CD95 (APO-1/Fas)-mediated apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Apaf-1 oligomerizes into biologically active approximately 700-kDa and inactive approximately 1.4-MDa apoptosome complexes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Caspase-9 can be activated without proteolytic processing

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Release of caspase-9 from mitochondria during neuronal apoptosis and cerebral ischemia

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Mass spectrometric identification of proteins released from mitochondria undergoing permeability transition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

The permeability transition pore complex: a target for apoptosis regulation by caspases and bcl-2-related proteins

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Genetic control of programmed cell death in the nematode C. elegans.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Yama/CPP32 beta, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Caenorhabditis elegans CED-9 protein is a bifunctional cell-death inhibitor

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Interaction of CED-4 with CED-3 and CED-9: a molecular framework for cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Bcl-XL interacts with Apaf-1 and inhibits Apaf-1-dependent caspase-9 activation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Pro-apoptotic apoptosis protease-activating factor 1 (Apaf-1) has a cytoplasmic localization distinct from Bcl-2 or Bcl-x(L).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

Direct interaction of the mitochondrial membrane protein carnitine palmitoyltransferase I with Bcl-2

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

L-carnitine prevents doxorubicin-induced apoptosis of cardiac myocytes: role of inhibition of ceramide generation.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2902010-X

10.1016/S0014-5793(00)02010-X

7 January 2021

Identifiers

DOI

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30 July 2017

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