(Q34100112)

31 July 2017

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title

The biology of the receptor for advanced glycation end products and its ligands (English)

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31 July 2017

A M Schmidt

1

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31 July 2017

S D Yan

series ordinal

2

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31 July 2017

S F Yan

series ordinal

3

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31 July 2017

D M Stern

series ordinal

4

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31 July 2017

language of work or name

English

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publication date

1 December 2000

1 reference

Biochimica et Biophysica Acta

31 July 2017

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31 July 2017

1498

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31 July 2017

2-3

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31 July 2017

99-111

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Activation of receptor for advanced glycation end products: a mechanism for chronic vascular dysfunction in diabetic vasculopathy and atherosclerosis

31 July 2017

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Receptor-dependent cell stress and amyloid accumulation in systemic amyloidosis

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

The dark side of glucose

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Human blood-brain barrier receptors for Alzheimer's amyloid-beta 1- 40. Asymmetrical binding, endocytosis, and transcytosis at the apical side of brain microvascular endothelial cell monolayer

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Advanced glycation endproducts interacting with their endothelial receptor induce expression of vascular cell adhesion molecule-1 (VCAM-1) in cultured human endothelial cells and in mice. A potential mechanism for the accelerated vasculopathy of dia

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Receptor-mediated endothelial cell dysfunction in diabetic vasculopathy. Soluble receptor for advanced glycation end products blocks hyperpermeability in diabetic rats

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Immunobiology of human vascular endothelium

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

The cellular and molecular mechanisms of diabetic complications

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Accumulation of Maillard reaction products in skin collagen in diabetes and aging

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

N(epsilon)-(carboxymethyl)lysine adducts of proteins are ligands for receptor for advanced glycation end products that activate cell signaling pathways and modulate gene expression

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

N epsilon-(carboxymethyl)lysine is a dominant advanced glycation end product (AGE) antigen in tissue proteins

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

The receptor for advanced glycation end products (RAGE) is a central mediator of the interaction of AGE-beta2microglobulin with human mononuclear phagocytes via an oxidant-sensitive pathway. Implications for the pathogenesis of dialysis-related amyl

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

beta 2-Microglobulin modified with advanced glycation end products is a major component of hemodialysis-associated amyloidosis

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Advanced glycation end products (AGEs) on the surface of diabetic erythrocytes bind to the vessel wall via a specific receptor inducing oxidant stress in the vasculature: a link between surface-associated AGEs and diabetic complications

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Increased vascular permeability in spontaneously diabetic BB/W rats and in rats with mild versus severe streptozocin-induced diabetes. Prevention by aldose reductase inhibitors and castration

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Suppression of accelerated diabetic atherosclerosis by the soluble receptor for advanced glycation endproducts

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Alzheimer's disease: a central role for amyloid

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Hereditary cerebral hemorrhage with amyloidosis in patients of Dutch origin is related to Alzheimer disease

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Amyloid-beta peptide-receptor for advanced glycation endproduct interaction elicits neuronal expression of macrophage-colony stimulating factor: a proinflammatory pathway in Alzheimer disease

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

A model for beta-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Different amyloidogenic peptides share a similar mechanism of neurotoxicity involving reactive oxygen species and calcium

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Free radicals and disruption of neuronal ion homeostasis in AD: a role for amyloid beta-peptide?

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Arresting amyloidosis in vivo using small-molecule anionic sulphonates or sulphates: implications for Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

HMG-1 as a late mediator of endotoxin lethality in mice

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Blockade of RAGE–amphoterin signalling suppresses tumour growth and metastases

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

Activation of the receptor for advanced glycation end products triggers a p21(ras)-dependent mitogen-activated protein kinase pathway regulated by oxidant stress.

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

A redox-triggered ras-effector interaction. Recruitment of phosphatidylinositol 3'-kinase to Ras by redox stress.

1 reference

https://api.crossref.org/works/10.1016%2FS0167-4889%2800%2900087-2

7 January 2021

10.1016/S0167-4889(00)00087-2

Identifiers

DOI

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31 July 2017

1 reference