(Q34139328)

31 July 2017

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title

VDAC regulation by the Bcl-2 family of proteins (English)

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31 July 2017

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Tsujimoto Y

1

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PubMed ID

11175254

31 July 2017

Shimizu S

series ordinal

2

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31 July 2017

1 December 2000

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Cell Death & Differentiation

31 July 2017

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31 July 2017

7

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31 July 2017

1174-1181

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31 July 2017

12

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https://scigraph.springernature.com/pub.10.1038/sj.cdd.4400780

31 July 2017

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Caspases: enemies within

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7 January 2021

DFF, a heterodimeric protein that functions downstream of caspase-3 to trigger DNA fragmentation during apoptosis

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7 January 2021

Cleavage of CAD inhibitor in CAD activation and DNA degradation during apoptosis

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7 January 2021

Acinus is a caspase-3-activated protein required for apoptotic chromatin condensation

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7 January 2021

The Bcl-2 protein family: arbiters of cell survival

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7 January 2021

Role of Bcl-2 family proteins in apoptosis: apoptosomes or mitochondria?

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Involvement of the bcl-2 gene in human follicular lymphoma

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Cloning the chromosomal breakpoint of t(14;18) human lymphomas: clustering around JH on chromosome 14 and near a transcriptional unit on 18.

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Nucleotide sequence of a t(14;18) chromosomal breakpoint in follicular lymphoma and demonstration of a breakpoint-cluster region near a transcriptionally active locus on chromosome 18.

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7 January 2021

Bcl-2 gene promotes haemopoietic cell survival and cooperates with c-myc to immortalize pre-B cells

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Structural basis of BFL-1 for its interaction with BAX and its anti-apoptotic action in mammalian and yeast cells

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Structure of Bcl-xL-Bak peptide complex: recognition between regulators of apoptosis

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

The protein bcl-2 alpha does not require membrane attachment, but two conserved domains to suppress apoptosis

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Structure-function analysis of Bcl-2 protein. Identification of conserved domains important for homodimerization with Bcl-2 and heterodimerization with Bax.

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Functional dissection of the human Bc12 protein: sequence requirements for inhibition of apoptosis

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Evidence for alpha-helical conformation of an essential N-terminal region in the human Bcl2 protein.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

The conserved N-terminal BH4 domain of Bcl-2 homologues is essential for inhibition of apoptosis and interaction with CED-4

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bcl-x(L) forms an ion channel in synthetic lipid membranes.

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Comparison of the ion channel characteristics of proapoptotic BAX and antiapoptotic BCL-2.

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Channel formation by antiapoptotic protein Bcl-2.

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Inhibition of Bax channel-forming activity by Bcl-2

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Ion channel activity of the BH3 only Bcl-2 family member, BID.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Mitochondria and apoptosis

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Molecular characterization of mitochondrial apoptosis-inducing factor

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bcl-XL interacts with Apaf-1 and inhibits Apaf-1-dependent caspase-9 activation

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Controlling cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bcl-2 family members do not inhibit apoptosis by binding the caspase activator Apaf-1

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Pro-apoptotic apoptosis protease-activating factor 1 (Apaf-1) has a cytoplasmic localization distinct from Bcl-2 or Bcl-x(L).

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bcl-2 and Bax function independently to regulate cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bax and Bcl-xL independently regulate apoptotic changes of yeast mitochondria that require VDAC but not adenine nucleotide translocator

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Investigation of bax-induced release of cytochrome c from yeast mitochondria permeability of mitochondrial membranes, role of VDAC and ATP requirement

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

A cytochrome c-GFP fusion is not released from mitochondria into the cytoplasm upon expression of Bax in yeast cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bcl-2 family: life-or-death switch

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bax, but not Bcl-xL, decreases the lifetime of planar phospholipid bilayer membranes at subnanomolar concentrations

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Mitochondrial permeability transition is a central coordinating event of apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

The mitochondrial permeability transition.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bax directly induces release of cytochrome c from isolated mitochondria

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

The mitochondrial death/life regulator in apoptosis and necrosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Functional consequences of the sustained or transient activation by Bax of the mitochondrial permeability transition pore

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Proapoptotic BH3-only Bcl-2 family members induce cytochrome c release, but not mitochondrial membrane potential loss, and do not directly modulate voltage-dependent anion channel activity

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

BAX-induced cell death may not require interleukin 1 beta-converting enzyme-like proteases

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

The overexpression of Bax produces cell death upon induction of the mitochondrial permeability transition

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Mitochondrial control of nuclear apoptosis.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Subcellular and submitochondrial mode of action of Bcl-2-like oncoproteins

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bcl-2 and Bax regulate the channel activity of the mitochondrial adenine nucleotide translocator

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Essential role of voltage-dependent anion channel in various forms of apoptosis in mammalian cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Biochemical and genetic analysis of the mitochondrial response of yeast to BAX and BCL-X(L)

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Electrophysiological study of a novel large pore formed by Bax and the voltage-dependent anion channel that is permeable to cytochrome c.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Mass spectrometric identification of proteins released from mitochondria undergoing permeability transition

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

X-ray and NMR structure of human Bcl-xL, an inhibitor of programmed cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bcl-2 family proteins as ion-channels

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

The pro-apoptotic proteins, Bid and Bax, cause a limited permeabilization of the mitochondrial outer membrane that is enhanced by cytosol

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Solution structure of BID, an intracellular amplifier of apoptotic signaling

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Solution structure of the proapoptotic molecule BID: a structural basis for apoptotic agonists and antagonists

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400780

7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4400780

1 reference

PubMed ID

11175254

Dimensions Publication ID

31 July 2017

0 references

1 reference

PubMed ID

11175254