(Q34162244)

31 July 2017

title

Mitochondria-targeted antioxidant prevents cardiac dysfunction induced by tafazzin gene knockdown in cardiac myocytes (English)

1 reference

31 July 2017

1 reference

Quan He

1

1 reference

31 July 2017

Nicole Harris

series ordinal

2

1 reference

31 July 2017

Jun Ren

series ordinal

3

1 reference

31 July 2017

Xianlin Han

series ordinal

4

1 reference

31 July 2017

publication date

27 August 2014

1 reference

Oxidative medicine and cellular longevity

31 July 2017

published in

1 reference

31 July 2017

volume

2014

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31 July 2017

page(s)

654198

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Creative Commons Attribution 3.0 Unported

31 July 2017

copyright license

start time

1 January 2014

1 reference

April 2022 Public Data File from Crossref

copyright status

copyrighted

1 reference

April 2022 Public Data File from Crossref

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A zebrafish model of human Barth syndrome reveals the essential role of tafazzin in cardiac development and function

5 July 2018

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Tafazzin knockdown interrupts cell cycle progression in cultured neonatal ventricular fibroblasts.

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Mitochondria-Targeted Antioxidant MitoQ 10 Improves Endothelial Function and Attenuates Cardiac Hypertrophy

21 January 2018

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Cardiolipin remodeling in diabetic heart

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Inhibitory effects of antioxidants on neonatal rat cardiac myocyte hypertrophy induced by tumor necrosis factor-alpha and angiotensin II

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Differing clinical courses and outcomes in two siblings with Barth syndrome and left ventricular noncompaction

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12 December 2020

Oxidative stress and heart failure

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12 December 2020

Oxidative stress causes heart failure with impaired mitochondrial respiration

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12 December 2020

Identifiers

DOI

10.1155/2014/654198

1 reference

31 July 2017

1 reference

31 July 2017

1 reference