(Q34175659)

31 July 2017

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title

Molecular biology of sodium channels and their role in cardiac arrhythmias (English)

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31 July 2017

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Grant AO

1

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31 July 2017

language of work or name

English

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publication date

1 March 2001

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American Journal of Medicine

31 July 2017

published in

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31 July 2017

volume

110

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31 July 2017

issue

4

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31 July 2017

page(s)

296-305

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A quantitative description of membrane current and its application to conduction and excitation in nerve

31 July 2017

cites work

1 reference

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Structure of the sodium channel pore revealed by serial cysteine mutagenesis.

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Calcium channel characteristics conferred on the sodium channel by single mutations

7 January 2021

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Ca2+ flux through promiscuous cardiac Na+ channels: slip-mode conductance.

7 January 2021

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Atrionatriuretic peptide transforms cardiac sodium channels into calcium-conducting channels

7 January 2021

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7 January 2021

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Structural parts involved in activation and inactivation of the sodium channel

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Na+ channels must deactivate to recover from inactivation

7 January 2021

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Accessory subunits and sodium channel inactivation

7 January 2021

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Evidence for the involvement of more than one mRNA species in controlling the inactivation process of rat and rabbit brain Na channels expressed in Xenopus oocytes

7 January 2021

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7 January 2021

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7 January 2021

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Sodium channel beta subunits mediate homophilic cell adhesion and recruit ankyrin to points of cell-cell contact

7 January 2021

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Novel LQT-3 mutation affects Na+ channel activity through interactions between alpha- and beta1-subunits

7 January 2021

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Local anesthetics: hydrophilic and hydrophobic pathways for the drug-receptor reaction

7 January 2021

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Time- and voltage-dependent interactions of antiarrhythmic drugs with cardiac sodium channels

7 January 2021

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7 January 2021

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Common molecular determinants of local anesthetic, antiarrhythmic, and anticonvulsant block of voltage-gated Na+ channels

7 January 2021

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Sodium channel selectivity filter regulates antiarrhythmic drug binding

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Long QT syndrome patients with mutations of the SCN5A and HERG genes have differential responses to Na+ channel blockade and to increases in heart rate. Implications for gene-specific therapy.

7 January 2021

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Molecular mechanism for an inherited cardiac arrhythmia

7 January 2021

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Phenotypic characterization of a novel long-QT syndrome mutation (R1623Q) in the cardiac sodium channel

7 January 2021

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A de novo missense mutation of human cardiac Na+ channel exhibiting novel molecular mechanisms of long QT syndrome

7 January 2021

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Linking a genetic defect to its cellular phenotype in a cardiac arrhythmia

7 January 2021

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A single Na(+) channel mutation causing both long-QT and Brugada syndromes

7 January 2021

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Two distinct congenital arrhythmias evoked by a multidysfunctional Na(+) channel

7 January 2021

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Enhanced Na(+) channel intermediate inactivation in Brugada syndrome

7 January 2021

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The Elusive Link Between LQT3 and Brugada Syndrome

7 January 2021

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10.1016/S0002-9343(00)00714-2

7 January 2021

Identifiers

DOI

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release_tmjv7ow3oreg3nhb2klu6oq4i4

31 July 2017

Fatcat ID

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Fatcat

https://api.fatcat.wiki/v0/release/tmjv7ow3oreg3nhb2klu6oq4i4

mapped directly with Wikidata item

24 November 2022

based on heuristic

1 reference