(Q34248491)

31 July 2017

1 reference

title

Disease-modifying anti-osteoarthritic drugs: current therapies and new prospects around protease inhibition (English)

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31 July 2017

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De Nanteuil G

1

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31 July 2017

Portevin B

series ordinal

2

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31 July 2017

Benoist A

series ordinal

3

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31 July 2017

publication date

1 January 2001

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31 July 2017

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31 July 2017

volume

56

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31 July 2017

issue

1-2

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31 July 2017

page(s)

107-112

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Mechanisms of joint destruction, pain and disability in osteoarthritis.

31 July 2017

cites work

1 reference

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Diacerhein and rhein reduce the ICE-induced IL-1beta and IL-18 activation in human osteoarthritic cartilage

7 January 2021

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Biochemical and histological changes in osteoarthritic synovial membrane

7 January 2021

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Acid, Neutral, and Alkaline Hydrolases in Arthritic Synovium

7 January 2021

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Augmented excretion of procathepsin L of a fos-transferred highly metastatic rat cell line

7 January 2021

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Comparative analysis of cathepsin l, cathepsin d, and collagenase messenger rna expression in synovial tissues of patients with rheumatoid arthritis and osteoarthritis, by in situ hybridization

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-827X%2801%2901027-8

Susceptibility of the cartilage collagens types II, IX and XI to degradation by the cysteine proteinases, cathepsins B and L

7 January 2021

1 reference

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Cathepsin B: an alternative protease for the generation of an aggrecan 'metalloproteinase' cleavage neoepitope

7 January 2021

1 reference

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Peptidyl fluoromethyl ketones as inhibitors of cathepsin B. Implication for treatment of rheumatoid arthritis

7 January 2021

1 reference

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Cysteine proteinase inhibitors decrease articular cartilage and bone destruction in chronic inflammatory arthritis

7 January 2021

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Inhibition of cartilage proteoglycan release by a specific inactivator of cathepsin B and an inhibitor of matrix metalloproteinases. Evidence for two converging pathways of chondrocyte-mediated proteoglycan degradation

7 January 2021

1 reference

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Cathepsin K, but not cathepsins B, L, or S, is abundantly expressed in human osteoclasts

7 January 2021

1 reference

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Interleukin-1beta-converting enzyme/caspase-1 in human osteoarthritic tissues: localization and role in the maturation of interleukin-1beta and interleukin-18

7 January 2021

1 reference

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Studies of thrombin-induced proteoglycan release in the degradation of human and bovine cartilage

7 January 2021

1 reference

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Synoviocytes from osteoarthritis and rheumatoid arthritis produce plasminogen activators and plasminogen activator inhibitor-1 and display u-PA receptors on their surface

7 January 2021

1 reference

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The synthesis of three 4-substituted benzo[b]thiophene-2-carboxamidines as potent and selective inhibitors of urokinase

7 January 2021

1 reference

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T-686, a novel inhibitor of plasminogen activator inhibitor-1, inhibits thrombosis without impairment of hemostasis in rats

7 January 2021

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The possible role of neutrophil proteinases in damage to articular cartilage. 1978.

7 January 2021

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Inhibition of cartilage degradation in rat collagen-induced arthritis but not adjuvant arthritis by the neutrophil elastase inhibitor MDL 101,146.

7 January 2021

1 reference

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Interaction between neutrophil-derived elastase and reactive oxygen species in cartilage degradation

7 January 2021

1 reference

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Dual inhibition of human leukocyte elastase and lipid peroxidation: in vitro and in vivo activities of azabicyclo[2.2.2]octane and perhydroindole derivatives

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-827X%2801%2901027-8

Selective enhancement of collagenase-mediated cleavage of resident type II collagen in cultured osteoarthritic cartilage and arrest with a synthetic inhibitor that spares collagenase 1 (matrix metalloproteinase 1)

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-827X%2801%2901027-8

Current pharmacological treatment of osteoarthritis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0014-827X%2801%2901027-8

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/S0014-827X(01)01027-8

1 reference

31 July 2017

1 reference