(Q34342629)

1 August 2017

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title

The role of NO in contact hypersensitivity (English)

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1 August 2017

Ross R

1

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1 August 2017

Reske-Kunz AB

series ordinal

2

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1 August 2017

publication date

1 August 2001

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International Immunopharmacology

1 August 2017

published in

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1 August 2017

volume

1

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1 August 2017

issue

8

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1 August 2017

page(s)

1469-1478

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STUDIES ON THE SENSITIZATION OF ANIMALS WITH SIMPLE CHEMICAL COMPOUNDS.

1 August 2017

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Studies on the sensitization of animals with simple chemical compounds. XII. The influence of excision of allergenic depots on onset of delayed hypersensitivity and tolerance

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L3T4 (CD4+) cells that mediate contact sensitivity to trinitrochlorobenzene express I-A determinants

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Immunoregulatory mechanisms involved in elicitation of allergic contact hypersensitivity

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Substance P and nitric oxide mediate would healing of ultraviolet photodamaged rat skin: evidence for an effect of nitric oxide on keratinocyte proliferation

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7 January 2021

Involvement of NO in contact hypersensitivity

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7 January 2021

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Dissection of antigenic and irritative effects of epicutaneously applied haptens in mice. Evidence that not the antigenic component but nonspecific proinflammatory effects of haptens determine the concentration-dependent elicitation of allergic cont

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7 January 2021

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7 January 2021

Groalpha-mediated recruitment of neutrophils is required for elicitation of contact hypersensitivity

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Interleukin 12 protects from a T helper type 1-mediated autoimmune disease, experimental autoimmune uveitis, through a mechanism involving interferon gamma, nitric oxide, and apoptosis.

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7 January 2021

Immune suppression by recombinant interleukin (rIL)-12 involves interferon gamma induction of nitric oxide synthase 2 (iNOS) activity: inhibitors of NO generation reveal the extent of rIL-12 vaccine adjuvant effect

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7 January 2021

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7 January 2021

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7 January 2021

10.1016/S1567-5769(01)00091-1

Identifiers

DOI

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1 August 2017

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