(Q34365223)

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At the limbic-motor interface: disconnection of basolateral amygdala from nucleus accumbens core and shell reveals dissociable components of incentive motivation

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scholarly article

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Europe PubMed Central

1 August 2017

At the limbic-motor interface: disconnection of basolateral amygdala from nucleus accumbens core and shell reveals dissociable components of incentive motivation (English)

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Europe PubMed Central

1 August 2017

nucleus accumbens

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based on heuristic

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author name string

Michael W Shiflett

1

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Europe PubMed Central

1 August 2017

Bernard W Balleine

2

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Europe PubMed Central

1 August 2017

language of work or name

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publication date

7 October 2010

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Europe PubMed Central

1 August 2017

European Journal of Neuroscience

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Europe PubMed Central

1 August 2017

32

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Europe PubMed Central

1 August 2017

10

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Europe PubMed Central

1 August 2017

1735-1743

1 reference

Europe PubMed Central

1 August 2017

Basolateral amygdala modulates terminal dopamine release in the nucleus accumbens and conditioned responding

1 reference

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Coherent gamma oscillations couple the amygdala and striatum during learning.

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Differential involvement of the basolateral amygdala and mediodorsal thalamus in instrumental action selection

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Selective activation of medial prefrontal-to-accumbens projection neurons by amygdala stimulation and Pavlovian conditioned stimuli

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Parallel incentive processing: an integrated view of amygdala function.

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Disconnection of the basolateral amygdala complex and nucleus accumbens impairs appetitive pavlovian second-order conditioned responses.

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Amygdala regulation of nucleus accumbens dopamine output is governed by the prefrontal cortex.

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Moment-to-moment tracking of state value in the amygdala.

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Orbitofrontal cortex mediates outcome encoding in Pavlovian but not instrumental conditioning.

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Lesions of the basolateral amygdala disrupt conditioning based on the retrieved representations of motivationally significant events.

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Double dissociation of basolateral and central amygdala lesions on the general and outcome-specific forms of pavlovian-instrumental transfer.

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Encoding predicted outcome and acquired value in orbitofrontal cortex during cue sampling depends upon input from basolateral amygdala.

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Lesions of the basolateral amygdala disrupt selective aspects of reinforcer representation in rats.

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Dopamine D1 and NMDA receptors mediate potentiation of basolateral amygdala-evoked firing of nucleus accumbens neurons.

1 reference

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The role of the nucleus accumbens in instrumental conditioning: Evidence of a functional dissociation between accumbens core and shell.

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Modulation of hippocampal and amygdalar-evoked activity of nucleus accumbens neurons by dopamine: cellular mechanisms of input selection.

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Evidence of a functional relationship between the nucleus accumbens shell and lateral hypothalamus subserving the control of feeding behavior.

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Neurotoxic lesions of basolateral, but not central, amygdala interfere with Pavlovian second-order conditioning and reinforcer devaluation effects.

1 reference

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Effects of ibotenic acid lesions of the nucleus accumbens on instrumental action.

1 reference

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27 October 2018

Identifiers

10.1111/J.1460-9568.2010.07439.X

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Europe PubMed Central

1 August 2017

1 reference

Europe PubMed Central

1 August 2017

1 reference

Europe PubMed Central

1 August 2017

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