(Q34407464)

2 August 2017

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title

Mechanisms of prion-induced modifications in membrane transport properties: implications for signal transduction and neurotoxicity (English)

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2 August 2017

0 references

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Kourie JI

1

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2 August 2017

publication date

1 October 2001

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Chemico-biological Interactions

2 August 2017

published in

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2 August 2017

volume

138

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2 August 2017

issue

1

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2 August 2017

page(s)

1-26

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2 August 2017

cites work

Prion Protein Biology

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Presence of prion protein in peripheral tissues of Libyan Jews with Creutzfeldt-Jakob disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Activation effects of a prion protein fragment [PrP-(106-126)] on human leucocytes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Pathologic conformations of prion proteins

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Intracellular calcium rise through L-type calcium channels, as molecular mechanism for prion protein fragment 106-126-induced astroglial proliferation.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Prion protein fragment 106-126 induces apoptotic cell death and impairment of L-type voltage-sensitive calcium channel activity in the GH3 cell line

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Channel formation by a neurotoxic prion protein fragment

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Prion peptide fragment PrP[106-126] forms distinct cation channel types.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Amyloid peptide channels: blockade by zinc and inhibition by Congo red (amyloid channel block).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Alzheimer's beta-amyloid, human islet amylin, and prion protein fragment evoke intracellular free calcium elevations by a common mechanism in a hypothalamic GnRH neuronal cell line

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Effect of flupirtine on Bcl-2 and glutathione level in neuronal cells treated in vitro with the prion protein fragment (PrP106-126)

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

A neurotoxic and gliotrophic fragment of the prion protein increases plasma membrane microviscosity.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

The 118-135 peptide of the human prion protein forms amyloid fibrils and induces liposome fusion.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Decreased receptor-mediated calcium response in prion-infected cells correlates with decreased membrane fluidity and IP3 release

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Signal transduction through prion protein

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

A Mutant Prion Protein Displays an Aberrant Membrane Association When Expressed in Cultured Cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Spongiform encephalopathies. Breech-birth prions

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Molecular characteristics of a protease-resistant, amyloidogenic and neurotoxic peptide homologous to residues 106-126 of the prion protein

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Scrapie prions alter receptor-mediated calcium responses in cultured cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Increase of intracellular free Ca2+ in microglia activated by prion protein fragment

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

High-dose exposure to systemic phosmet insecticide modifies the phosphatidylinositol anchor on the prion protein: the origins of new variant transmissible spongiform encephalopathies?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Activation of microglial cells by PrP and beta-amyloid fragments raises intracellular calcium through L-type voltage sensitive calcium channels

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Enhancement of (45)Ca(2+) influx and voltage-dependent Ca(2+) channel activity by beta-amyloid-(1-40) in rat cortical synaptosomes and cultured cortical neurons. Modulation by the proinflammatory cytokine interleukin-1beta

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Neurotoxicity of A beta amyloid protein in vitro is not altered by calcium channel blockade

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Amyloid beta protein-(1-42) forms calcium-permeable, Zn2+-sensitive channel

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Regulation of intracellular free calcium levels by the cellular prion protein

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Spongy state (status spongiosus) and inhibition of Na, K-ATPase: A pathogenetic theory

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Prion protein is necessary for normal synaptic function

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Hippocampal slices from prion protein null mice: disrupted Ca(2+)-activated K+ currents.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Intrinsic physiological and morphological properties of principal cells of the hippocampus and neocortex in hamsters infected with scrapie

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Patch-clamp analysis of synaptic transmission to cerebellar purkinje cells of prion protein knockout mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Mice deficient for prion protein exhibit normal neuronal excitability and synaptic transmission in the hippocampus

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

B lymphocytes and neuroinvasion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Antioxidant activity related to copper binding of native prion protein

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Prion protein-deficient neurons reveal lower glutathione reductase activity and increased susceptibility to hydrogen peroxide toxicity

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Strain-specific prion-protein conformation determined by metal ions

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Copper binding to the prion protein: structural implications of four identical cooperative binding sites

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Normal prion protein has an activity like that of superoxide dismutase.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Prion protein expression aids cellular uptake and veratridine-induced release of copper

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Copper refolding of prion protein.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Selective oxidation of methionine residues in prion proteins

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Interaction of reactive oxygen species with ion transport mechanisms.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Microglial cells respond to amyloidogenic PrP peptide by the production of inflammatory cytokines

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

PrP and beta-amyloid fragments activate different neurotoxic mechanisms in cultured mouse cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Neurotoxicity of a prion protein fragment

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Theoretical analysis of the implication of PrP in neuronal death during transmissible subacute spongiform encephalopathies: hypothesis of a PrP oligomeric channel.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Tetracycline affects abnormal properties of synthetic PrP peptides and PrP(Sc) in vitro.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Multiple folding pathways for heterologously expressed human prion protein

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Spontaneous deamidation and isomerization of Asn108 in prion peptide 106-126 and in full-length prion protein

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

In vivo cytotoxicity of the prion protein fragment 106-126

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Neurotoxicity of prion peptide 106-126 not confirmed

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Recombinant scrapie-like prion protein of 106 amino acids is soluble

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

The hydrophobic core sequence modulates the neurotoxic and secondary structure properties of the prion peptide 106-126.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

A transmembrane form of the prion protein in neurodegenerative disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Structural changes in a hydrophobic domain of the prion protein induced by hydration and by ala-->Val and pro-->Leu substitutions

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Transmissible and genetic prion diseases share a common pathway of neurodegeneration

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Structural mobility of the human prion protein probed by backbone hydrogen exchange

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

No propagation of prions in mice devoid of PrP.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Neurotoxicity of the Putative Transmembrane Domain of the Prion Protein

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Evidence for the conformation of the pathologic isoform of the prion protein enciphering and propagating prion diversity

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Structural neurology: are seeds at the root of neuronal degeneration?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Reported channel formation by prion protein fragment 106-126 in planar lipid bilayers cannot be reproduced

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

Infectivity and host responses in Creutzfeldt-Jakob disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0009-2797%2801%2900228-9

10.1016/S0009-2797(01)00228-9

7 January 2021

Identifiers

DOI

1 reference

2 August 2017

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