(Q34446495)

2 August 2017

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title

Polyglutamine expansion neurodegenerative disease (English)

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2 August 2017

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Fischbeck KH

1

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2 August 2017

publication date

1 October 2001

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2 August 2017

published in

Brain Research Bulletin

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2 August 2017

volume

56

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2 August 2017

issue

3-4

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2 August 2017

page(s)

161-163

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Expression of expanded repeat androgen receptor produces neurologic disease in transgenic mice.

2 August 2017

cites work

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SCA1 transgenic mice: A model for neurodegeneration caused by an expanded CAG trinucleotide repeat

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Genetic suppression of polyglutamine toxicity in Drosophila

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Progressive proximal spinal and bulbar muscular atrophy of late onset. A sex-linked recessive trait

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Ataxin-1 nuclear localization and aggregation: role in polyglutamine-induced disease in SCA1 transgenic mice.

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7 January 2021

Androgen receptor gene mutations in X-linked spinal and bulbar muscular atrophy

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7 January 2021

Meiotic stability and genotype-phenotype correlation of the trinucleotide repeat in X-linked spinal and bulbar muscular atrophy

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7 January 2021

CREB-binding protein sequestration by expanded polyglutamine

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7 January 2021

Cleavage, aggregation and toxicity of the expanded androgen receptor in spinal and bulbar muscular atrophy

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7 January 2021

Trinucleotide repeats in neurogenetic disorders

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Intranuclear inclusions of expanded polyglutamine protein in spinocerebellar ataxia type 3.

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7 January 2021

Huntingtin acts in the nucleus to induce apoptosis but death does not correlate with the formation of intranuclear inclusions.

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7 January 2021

X-linked recessive bulbospinal neuronopathy. A clinicopathological study

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The Huntington's disease protein interacts with p53 and CREB-binding protein and represses transcription

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7 January 2021

Expanded polyglutamine protein forms nuclear inclusions and causes neural degeneration in Drosophila

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Reversal of neuropathology and motor dysfunction in a conditional model of Huntington's disease

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10.1016/S0361-9230(01)00577-9

Identifiers

DOI

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2 August 2017

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