(Q34486849)

2 August 2017

Autoimmune T cells protect neurons from secondary degeneration after central nervous system axotomy (English)

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2 August 2017

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Moalem G

1

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2 August 2017

Leibowitz-Amit R

2

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2 August 2017

Yoles E

3

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2 August 2017

Mor F

4

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2 August 2017

Cohen IR

5

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2 August 2017

Schwartz M

6

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2 August 2017

language of work or name

English

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publication date

1 January 1999

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2 August 2017

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2 August 2017

5

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2 August 2017

49-55

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2 August 2017

1

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https://scigraph.springernature.com/pub.10.1038/4734

2 August 2017

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Dynamic changes in local cerebral glucose utilization following cerebral concussion in rats: evidence of a hyper- and subsequent hypometabolic state

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Diffuse prolonged depression of cerebral oxidative metabolism following concussive brain injury in the rat: a cytochrome oxidase histochemistry study

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NMDA-receptor antagonist protects neurons from secondary degeneration after partial optic nerve crush.

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Degeneration of spared axons following partial white matter lesion: implications for optic nerve neuropathies.

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7 January 2021

T lymphocyte lines producing or vaccinating against autoimmune encephalomyelitis (EAE). Functional activation induces peanut agglutinin receptors and accumulation in the brain and thymus of line cells

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Concept of autoimmunity following spinal cord injury: possible roles for T lymphocytes in the traumatized central nervous system

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Rat CNS white matter, but not gray matter, is nonpermissive for neuronal cell adhesion and fiber outgrowth

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Cross talk between the immune system and the nervous system in response to injury: implications for regeneration.

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The T lymphocyte in experimental allergic encephalomyelitis

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Immunocomplexes in rat and rabbit spinal cord after injury

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Autoimmunity to Chaperonins in the Pathogenesis of Arthritis and Diabetes

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T cells in the lesion of experimental autoimmune encephalomyelitis. Enrichment for reactivities to myelin basic protein and to heat shock proteins

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7 January 2021

Brain cooling during transient focal ischemia provides complete neuroprotection

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Immunospecific inhibition of nerve conduction by T lymphocytes reactive to basic protein of myelin

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Immunologically induced electrophysiological dysfunction: implications for inflammatory diseases of the CNS and PNS.

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The distribution of inflammatory demyelinated lesions in the central nervous system of rats with antibody-augmented demyelinating experimental allergic encephalomyelitis.

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Expression of nerve growth factor and nerve growth factor receptor tyrosine kinase Trk in activated CD4-positive T-cell clones

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Human CD4+ T cell clones produce and release nerve growth factor and express high-affinity nerve growth factor receptors

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The cognitive paradigm and the immunological homunculus

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Anti-idiotypic network induced by T cell vaccination against experimental autoimmune encephalomyelitis

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Tolerance, danger, and the extended family

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Basic encephalitogenic protein: A simplified purification on sulphoethyl-sephadex

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Vaccination against autoimmune mouse diabetes with a T-cell epitope of the human 65-kDa heat shock protein.

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7 January 2021

Shifts in the epitopes of myelin basic protein recognized by Lewis rat T cells before, during, and after the induction of experimental autoimmune encephalomyelitis

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A monoclonal antibody to a constant determinant of the rat T cell antigen receptor that induces T cell activation. Differential reactivity with subsets of immature and mature T lymphocytes

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HU-211, a nonpsychotropic cannabinoid, produces short- and long-term neuroprotection after optic nerve axotomy

1 reference

7 January 2021

Identifiers

DOI

10.1038/4734

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Dimensions Publication ID

2 August 2017

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