(Q35077031)

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Oncolytic vesicular stomatitis virus induces apoptosis in U87 glioblastoma cells by a type II death receptor mechanism and induces cell death and tumor clearance in vivo

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scholarly article

1 reference

Europe PubMed Central

6 August 2017

Oncolytic vesicular stomatitis virus induces apoptosis in U87 glioblastoma cells by a type II death receptor mechanism and induces cell death and tumor clearance in vivo (English)

1 reference

Europe PubMed Central

6 August 2017

oncolytic virus

1 reference

based on heuristic

inferred from title

1 reference

based on heuristic

inferred from title

vesicular stomatitis

1 reference

based on heuristic

inferred from title

author name string

Zachary D Cary

1

1 reference

Europe PubMed Central

6 August 2017

Mark C Willingham

2

1 reference

Europe PubMed Central

6 August 2017

Douglas S Lyles

3

1 reference

Europe PubMed Central

6 August 2017

language of work or name

0 references

publication date

30 March 2011

1 reference

Europe PubMed Central

6 August 2017

Journal of Virology

1 reference

Europe PubMed Central

6 August 2017

85

1 reference

Europe PubMed Central

6 August 2017

12

1 reference

Europe PubMed Central

6 August 2017

5708-5717

1 reference

Europe PubMed Central

6 August 2017

describes a project that uses

1 reference

Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC3126314/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

Susceptibility of breast cancer cells to an oncolytic matrix (M) protein mutant of vesicular stomatitis virus

1 reference

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1 reference

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Some attenuated variants of vesicular stomatitis virus show enhanced oncolytic activity against human glioblastoma cells relative to normal brain cells

1 reference

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Peripheral immunization blocks lethal actions of vesicular stomatitis virus within the brain.

1 reference

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XIAP discriminates between type I and type II FAS-induced apoptosis

1 reference

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Vesicular stomatitis virus induces apoptosis primarily through Bak rather than Bax by inactivating Mcl-1 and Bcl-XL

1 reference

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Targeting the apoptotic pathway with BCL-2 inhibitors sensitizes primary chronic lymphocytic leukemia cells to vesicular stomatitis virus-induced oncolysis

1 reference

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1 reference

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1 reference

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Oncolytic vesicular stomatitis virus induces apoptosis via signaling through PKR, Fas, and Daxx

1 reference

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Variable deficiencies in the interferon response enhance susceptibility to vesicular stomatitis virus oncolytic actions in glioblastoma cells but not in normal human glial cells

1 reference

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Effects of intravenously administered recombinant vesicular stomatitis virus (VSV(deltaM51)) on multifocal and invasive gliomas.

1 reference

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Phase I/II trial of intravenous NDV-HUJ oncolytic virus in recurrent glioblastoma multiforme

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Targeting human glioblastoma cells: comparison of nine viruses with oncolytic potential

1 reference

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Vesicular stomatitis viruses expressing wild-type or mutant M proteins activate apoptosis through distinct pathways

1 reference

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Sensitivity of prostate tumors to wild type and M protein mutant vesicular stomatitis viruses

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VIAF, a conserved inhibitor of apoptosis (IAP)-interacting factor that modulates caspase activation

1 reference

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Upstream regulatory role for XIAP in receptor-mediated apoptosis

1 reference

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1 reference

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Syncytia induction enhances the oncolytic potential of vesicular stomatitis virus in virotherapy for cancer

1 reference

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1 reference

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Contrasting effects of matrix protein on apoptosis in HeLa and BHK cells infected with vesicular stomatitis virus are due to inhibition of host gene expression

1 reference

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1 reference

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Matrix protein and another viral component contribute to induction of apoptosis in cells infected with vesicular stomatitis virus

1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3126314

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1 reference

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1 reference

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1128/JVI.02393-10

1 reference

Europe PubMed Central

6 August 2017

1 reference

Europe PubMed Central

6 August 2017

1 reference

Europe PubMed Central

6 August 2017

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