(Q35084876)

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Inhibition of macrophage nuclear factor-kappaB leads to a dominant anti-inflammatory phenotype that attenuates glomerular inflammation in vivo.

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15972949%20AND%20SRC:MED&resulttype=core&format=json

13 January 2020

Inhibition of macrophage nuclear factor-kappaB leads to a dominant anti-inflammatory phenotype that attenuates glomerular inflammation in vivo (English)

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13 January 2020

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Heather M Wilson

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13 January 2020

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Salah Chettibi

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13 January 2020

Christian Jobin

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13 January 2020

David Walbaum

4

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13 January 2020

Andrew J Rees

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13 January 2020

David C Kluth

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13 January 2020

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1 July 2005

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13 January 2020

The American Journal of Pathology

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13 January 2020

167

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13 January 2020

1

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13 January 2020

27-37

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13 January 2020

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1 reference

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Alternative activation of macrophages

1 reference

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Regulation of c-myc expression by IFN-gamma through Stat1-dependent and -independent pathways.

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Specific NF-kappaB blockade selectively inhibits tumour necrosis factor-alpha-induced COX-2 but not constitutive COX-1 gene expression in HT-29 cells

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TNF- and cancer therapy-induced apoptosis: potentiation by inhibition of NF-kappaB

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Glomerular expression of interleukin-1 receptor antagonist and interleukin-1 beta genes in antibody-mediated glomerulonephritis

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Evidence for a pathogenic role of a cell-mediated immune mechanism in experimental glomerulonephritis

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1 reference

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IL-12 directs severe renal injury, crescent formation and Th1 responses in murine glomerulonephritis

1 reference

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12 December 2020

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10.1016/S0002-9440(10)62950-1

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Europe PubMed Central

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13 January 2020

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Europe PubMed Central

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13 January 2020

PubMed publication ID

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Europe PubMed Central

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13 January 2020

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