(Q35091380)

6 August 2017

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title

Death receptors leave a caspase footprint that Smacs of XIAP. (English)

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6 August 2017

Bratton SB

1

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6 August 2017

Cohen GM

series ordinal

2

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6 August 2017

publication date

1 January 2003

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Cell Death & Differentiation

6 August 2017

published in

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6 August 2017

volume

10

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6 August 2017

issue

1

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6 August 2017

page(s)

4-6

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The CD95 (APO-1/Fas) and the TRAIL (APO-2L) apoptosis systems

6 August 2017

cites work

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401176

7 January 2021

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401176

7 January 2021

Activation of Fas by FasL induces apoptosis by a mechanism that cannot be blocked by Bcl-2 or Bcl-x(L)

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401176

7 January 2021

Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401176

7 January 2021

Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401176

7 January 2021

Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401176

7 January 2021

Structural and biochemical basis of apoptotic activation by Smac/DIABLO

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401176

7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4401176

1 reference

6 August 2017

1 reference