(Q36098835)

English

Bortezomib induces apoptosis of Epstein-Barr virus (EBV)-transformed B cells and prolongs survival of mice inoculated with EBV-transformed B cells

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scholarly article

1 reference

Europe PubMed Central

12 August 2017

Bortezomib induces apoptosis of Epstein-Barr virus (EBV)-transformed B cells and prolongs survival of mice inoculated with EBV-transformed B cells (English)

1 reference

Europe PubMed Central

12 August 2017

Epstein–Barr virus

0 references

apoptotic process

1 reference

based on heuristic

inferred from title

author name string

Ping Zou

1

1 reference

Europe PubMed Central

12 August 2017

Junichi Kawada

2

1 reference

Europe PubMed Central

12 August 2017

Lesley Pesnicak

3

1 reference

Europe PubMed Central

12 August 2017

Jeffrey I Cohen

4

1 reference

Europe PubMed Central

12 August 2017

language of work or name

0 references

publication date

11 July 2007

1 reference

Europe PubMed Central

12 August 2017

Journal of Virology

1 reference

Europe PubMed Central

12 August 2017

81

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Europe PubMed Central

12 August 2017

18

1 reference

Europe PubMed Central

12 August 2017

10029-10036

1 reference

Europe PubMed Central

12 August 2017

describes a project that uses

1 reference

Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC2045383/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

Bortezomib: proteasome inhibition as an effective anticancer therapy

1 reference

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NF-kappaB is essential for the progression of KSHV- and EBV-infected lymphomas in vivo

1 reference

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Bortezomib, a novel proteasome inhibitor, in the treatment of hematologic malignancies

1 reference

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Role of NF-kappa B in cell survival and transcription of latent membrane protein 1-expressing or Epstein-Barr virus latency III-infected cells

1 reference

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Simvastatin induces apoptosis of Epstein-Barr virus (EBV)-transformed lymphoblastoid cell lines and delays development of EBV lymphomas.

1 reference

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Epstein-Barr virus latent infection membrane protein 1 TRAF-binding site induces NIK/IKK alpha-dependent noncanonical NF-kappaB activation

1 reference

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1 reference

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1 reference

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1 reference

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NF-kappaB-inducing kinase regulates the processing of NF-kappaB2 p100

1 reference

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Epstein-Barr virus infection

1 reference

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NF-kappa B inhibition causes spontaneous apoptosis in Epstein-Barr virus-transformed lymphoblastoid cells

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The Epstein-Barr virus oncoprotein latent membrane protein 1 engages the tumor necrosis factor receptor-associated proteins TRADD and receptor-interacting protein (RIP) but does not induce apoptosis or require RIP for NF-kappaB activation

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Role of the TRAF binding site and NF-kappaB activation in Epstein-Barr virus latent membrane protein 1-induced cell gene expression

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Association of TRAF1, TRAF2, and TRAF3 with an Epstein-Barr virus LMP1 domain important for B-lymphocyte transformation: role in NF-kappaB activation

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1 reference

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based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1128/JVI.02241-06

1 reference

Europe PubMed Central

12 August 2017

1 reference

Europe PubMed Central

12 August 2017

1 reference

Europe PubMed Central

12 August 2017

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