(Q36142000)

English

Necrotic myocardial cells release damage-associated molecular patterns that provoke fibroblast activation in vitro and trigger myocardial inflammation and fibrosis in vivo.

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scholarly article

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16 February 2020

Necrotic myocardial cells release damage-associated molecular patterns that provoke fibroblast activation in vitro and trigger myocardial inflammation and fibrosis in vivo (English)

1 reference

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16 February 2020

3

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16 February 2020

author name string

Weili Zhang

1

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16 February 2020

Kory J Lavine

2

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16 February 2020

Sarah A Evans

4

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16 February 2020

Carla J Weinheimer

5

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16 February 2020

Philip M Barger

6

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16 February 2020

Douglas L Mann

7

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16 February 2020

publication date

2 June 2015

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16 February 2020

Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease

1 reference

Europe PubMed Central

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16 February 2020

4

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16 February 2020

6

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16 February 2020

e001993

1 reference

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16 February 2020

Beyond tissue injury-damage-associated molecular patterns, toll-like receptors, and inflammasomes also drive regeneration and fibrosis

1 reference

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Innate immunity and cardiomyocytes in ischemic heart disease

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Emerging role of high-mobility group box 1 (HMGB1) in liver diseases

1 reference

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2 October 2017

A necrotic stimulus is required to maximize matrix-mediated myogenesis in mice

1 reference

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2 October 2017

Injected matrix stimulates myogenesis and regeneration of mouse skeletal muscle after ischaemic injury.

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Toll-like receptors in liver fibrosis: cellular crosstalk and mechanisms

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High mobility group B1 impairs hepatocyte regeneration in acetaminophen hepatotoxicity

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HMGB1: a multifunctional alarmin driving autoimmune and inflammatory disease

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Redox modification of cysteine residues regulates the cytokine activity of high mobility group box-1 (HMGB1)

1 reference

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2 October 2017

The development of myocardial fibrosis in transgenic mice with targeted overexpression of tumor necrosis factor requires mast cell-fibroblast interactions

1 reference

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2 October 2017

High mobility group box-1 induces migration of vascular smooth muscle cells via TLR4-dependent PI3K/Akt pathway activation

1 reference

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2 October 2017

Innate immune signaling in cardiac ischemia

1 reference

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2 October 2017

Oxidative modifications of DAMPs suppress inflammation: the case for S100A8 and S100A9.

1 reference

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2 October 2017

Methylation determines fibroblast activation and fibrogenesis in the kidney

1 reference

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2 October 2017

The sterile inflammatory response

1 reference

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2 October 2017

Pattern recognition receptors and inflammation

1 reference

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2 October 2017

How dying cells alert the immune system to danger

1 reference

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2 October 2017

The mechanistic basis of infarct healing.

1 reference

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2 October 2017

Cutting edge: extracellular high mobility group box-1 protein is a proangiogenic cytokine

1 reference

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2 October 2017

The dying stem cell hypothesis: immune modulation as a novel mechanism for progenitor cell therapy in cardiac muscle

1 reference

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2 October 2017

Hydrophobicity: an ancient damage-associated molecular pattern that initiates innate immune responses.

1 reference

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2 October 2017

Extracellular HMGB1, a signal of tissue damage, induces mesoangioblast migration and proliferation

1 reference

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2 October 2017

Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretion

1 reference

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2 October 2017

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

1 reference

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2 October 2017

Differential roles of TLR2 and TLR4 in recognition of gram-negative and gram-positive bacterial cell wall components

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2 October 2017

Tolerance, danger, and the extended family

1 reference

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2 October 2017

Approaching the asymptote? Evolution and revolution in immunology

1 reference

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2 October 2017

Tumor necrosis factor receptor-associated factor 2 signaling provokes adverse cardiac remodeling in the adult mammalian heart

1 reference

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2 July 2018

Hmgb1 promotes wound healing of 3T3 mouse fibroblasts via RAGE-dependent ERK1/2 activation.

1 reference

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2 July 2018

The alarmin cytokine, high mobility group box 1, is produced by viable cardiomyocytes and mediates the lipopolysaccharide-induced myocardial dysfunction via a TLR4/phosphatidylinositol 3-kinase gamma pathway.

1 reference

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2 July 2018

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23 September 2018

What can tubular progenitor cultures teach us about kidney regeneration?

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23 September 2018

High-mobility group box 1 release and redox regulation accompany regeneration and remodeling of skeletal muscle.

1 reference

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23 September 2018

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1 reference

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23 September 2018

Exogenous High-Mobility Group Box 1 Protein Induces Myocardial Regeneration After Infarction via Enhanced Cardiac C-Kit+ Cell Proliferation and Differentiation

1 reference

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23 September 2018

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1 reference

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Activation of CD4+ T lymphocytes improves wound healing and survival after experimental myocardial infarction in mice

1 reference

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12 December 2020

based on heuristic

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Multiple Effects of High Mobility Group Box Protein 1 in Skeletal Muscle Regeneration

1 reference

https://pubmed.ncbi.nlm.nih.gov/26037082

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Introduction: the role of innate immunity in the adaptive immune response

1 reference

https://pubmed.ncbi.nlm.nih.gov/26037082

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1161/JAHA.115.001993

1 reference

Europe PubMed Central

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16 February 2020

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26037082%20AND%20SRC:MED&resulttype=core&format=json

16 February 2020

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26037082%20AND%20SRC:MED&resulttype=core&format=json

16 February 2020

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