(Q36312494)

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Neuronal Wiskott-Aldrich syndrome protein (N-WASP) is critical for formation of α-smooth muscle actin filaments during myofibroblast differentiation

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Neuronal Wiskott-Aldrich syndrome protein (N-WASP) is critical for formation of α-smooth muscle actin filaments during myofibroblast differentiation (English)

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

1 reference

based on heuristic

inferred from title

Wiskott-Aldrich syndrome

1 reference

based on heuristic

inferred from title

author name string

Guo-Qiang Cai

1

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Chu-Fang Chou

2

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Meng Hu

3

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Anni Zheng

4

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Louis F Reichardt

5

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Jun-Lin Guan

6

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Haotian Fang

7

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Tracy R Luckhardt

8

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Yong Zhou

9

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Victor J Thannickal

10

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Qiang Ding

11

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

publication date

10 August 2012

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

American Journal of Physiology - Lung Cellular and Molecular Physiology

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

303

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

8

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

L692-702

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Matrix stiffness-induced myofibroblast differentiation is mediated by intrinsic mechanotransduction

1 reference

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New insights into the pathogenesis and treatment of idiopathic pulmonary fibrosis

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Downregulation of FAK-related non-kinase mediates the migratory phenotype of human fibrotic lung fibroblasts

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Force-induced myofibroblast differentiation through collagen receptors is dependent on mammalian diaphanous (mDia).

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1 reference

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Actin filament nucleation and elongation factors--structure-function relationships

1 reference

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Wash functions downstream of Rho and links linear and branched actin nucleation factors

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Myofibroblast contraction activates latent TGF-beta1 from the extracellular matrix

1 reference

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The myofibroblast: one function, multiple origins

1 reference

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The WASP-WAVE protein network: connecting the membrane to the cytoskeleton

1 reference

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Combinatorial activation of FAK and AKT by transforming growth factor-beta1 confers an anoikis-resistant phenotype to myofibroblasts

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Transforming growth factor beta: a central modulator of pulmonary and airway inflammation and fibrosis

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Ligation of protease-activated receptor 1 enhances alpha(v)beta6 integrin-dependent TGF-beta activation and promotes acute lung injury.

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Interaction of N-WASP with hnRNPK and its role in filopodia formation and cell spreading

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Tissue repair, contraction, and the myofibroblast

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Cortactin regulates cell migration through activation of N-WASP

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p27Kip1 and cyclin D1 are necessary for focal adhesion kinase regulation of cell cycle progression in glioblastoma cells propagated in vitro and in vivo in the scid mouse brain

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Regulation of WASP/WAVE proteins: making a long story short

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FAK and paxillin: regulators of N-cadherin adhesion and inhibitors of cell migration?

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Mechanisms of pulmonary fibrosis

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Focal adhesion kinase regulation of N-WASP subcellular localization and function

1 reference

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FAK deficiency in cells contributing to the basal lamina results in cortical abnormalities resembling congenital muscular dystrophies

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Alpha-smooth muscle actin is crucial for focal adhesion maturation in myofibroblasts.

1 reference

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Focal adhesion kinase: the first ten years

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Cellular motility driven by assembly and disassembly of actin filaments

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The myofibroblast in pulmonary fibrosis

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The NH2-terminal peptide of alpha-smooth muscle actin inhibits force generation by the myofibroblast in vitro and in vivo

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N-WASP activation by a beta1-integrin-dependent mechanism supports PI3K-independent chemotaxis stimulated by urokinase-type plasminogen activator

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Alpha-smooth muscle actin expression upregulates fibroblast contractile activity

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WASP and WAVE family proteins: key molecules for rapid rearrangement of cortical actin filaments and cell movement

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Identifiers

10.1152/AJPLUNG.00390.2011

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

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