(Q36462824)

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Endoplasmic reticulum stress and unfolded protein response in Atm-deficient thymocytes and thymic lymphoma cells are attributable to oxidative stress

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

Endoplasmic reticulum stress and unfolded protein response in Atm-deficient thymocytes and thymic lymphoma cells are attributable to oxidative stress (English)

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

endoplasmic reticulum

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author name string

Mingshan Yan

1

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

Jianjun Shen

2

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

Maria D Person

3

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

Xianghong Kuang

4

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

William S Lynn

5

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

Daphne Atlas

6

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

Paul K Y Wong

7

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Europe PubMed Central

PMC publication ID

14 August 2017

publication date

1 February 2008

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Europe PubMed Central

PMC publication ID

14 August 2017

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

10

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Europe PubMed Central

PMC publication ID

14 August 2017

2

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

160-167

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage

1 reference

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Oxidative stress and cancer: have we moved forward?

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The endoplasmic reticulum: folding, calcium homeostasis, signaling, and redox control

1 reference

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ER stress and cancer

1 reference

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ATM controls c-Myc and DNA synthesis during postnatal thymocyte development through regulation of redox state.

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Control of Atm-/- thymic lymphoma cell proliferation in vitro and in vivo by dexamethasone

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Role of reactive oxygen species in skin carcinogenesis

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PERK-dependent activation of Nrf2 contributes to redox homeostasis and cell survival following endoplasmic reticulum stress

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DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation

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ATM deficiency and oxidative stress: a new dimension of defective response to DNA damage

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Down-regulation of topoisomerase II alpha is caused by up-regulation of GRP78.

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ER stress regulation of ATF6 localization by dissociation of BiP/GRP78 binding and unmasking of Golgi localization signals

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XBP1 mRNA is induced by ATF6 and spliced by IRE1 in response to ER stress to produce a highly active transcription factor

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21 September 2018

Identifiers

10.1593/NEO.07935

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

14 August 2017

ResearchGate publication ID

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