(Q36617105)

15 August 2017

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title

IkappaB-zeta: an inducible regulator of nuclear factor-kappaB. (English)

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15 August 2017

Tatsushi Muta

1

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15 August 2017

publication date

1 January 2006

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15 August 2017

published in

Vitamins and Hormones

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15 August 2017

volume

74

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15 August 2017

page(s)

301-316

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An AU-rich sequence element (UUUN[A/U]U) downstream of the edited C in apolipoprotein B mRNA is a high-affinity binding site for Apobec-1: binding of Apobec-1 to this motif in the 3' untranslated region of c-myc increases mRNA stability

15 August 2017

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7 January 2021

Essential roles for NF-kappa B and a Toll/IL-1 receptor domain-specific signal(s) in the induction of I kappa B-zeta

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7 January 2021

Chronic cicatrizing conjunctivitis

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Peptide-induced negative selection of thymocytes activates transcription of an NF-kappa B inhibitor

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7 January 2021

Isolation of a novel interleukin-1-inducible nuclear protein bearing ankyrin-repeat motifs

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7 January 2021

The nuclear IkappaB protein IkappaBNS selectively inhibits lipopolysaccharide-induced IL-6 production in macrophages of the colonic lamina propria

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7 January 2021

I kappa B gamma, a 70 kd protein identical to the C-terminal half of p110 NF-kappa B: a new member of the I kappa B family

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7 January 2021

TAK1 mediates an activation signal from toll-like receptor(s) to nuclear factor-kappaB in lipopolysaccharide-stimulated macrophages

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7 January 2021

Transcriptional regulation of the MAIL gene in LPS-stimulated RAW264 mouse macrophages

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Approaching the asymptote? Evolution and revolution in immunology

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Innate immune recognition

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Phosphorylation meets ubiquitination: the control of NF-[kappa]B activity

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7 January 2021

MAIL, a novel nuclear I kappa B protein that potentiates LPS-induced IL-6 production

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Bacterial lipopolysaccharide induces mRNA expression of an IkappaB MAIL through toll-like receptor 4.

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Bacterial lipopolysaccharide-induced expression of the IkappaB protein MAIL in B-lymphocytes and macrophages

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7 January 2021

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Innate immune induction of the adaptive immune response

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mRNA stability in eukaryotes

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7 January 2021

Positive and negative regulation of nuclear factor-kappaB-mediated transcription by IkappaB-zeta, an inducible nuclear protein

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7 January 2021

Identification of HuR as a protein implicated in AUUUA-mediated mRNA decay

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7 January 2021

The kinase TAK1 can activate the NIK-I kappaB as well as the MAP kinase cascade in the IL-1 signalling pathway

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The candidate proto-oncogene bcl-3 is related to genes implicated in cell lineage determination and cell cycle control

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7 January 2021

TICAM-1, an adaptor molecule that participates in Toll-like receptor 3-mediated interferon-beta induction

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7 January 2021

Genomic organization, chromosomal localization, and promoter analysis of the mouse Mail gene

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7 January 2021

Targeted disruption of MAIL, a nuclear IkappaB protein, leads to severe atopic dermatitis-like disease

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7 January 2021

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7 January 2021

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7 January 2021

Recognition of gram-negative bacteria and endotoxin by the innate immune system

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7 January 2021

Tumor necrosis factor receptor and Fas signaling mechanisms.

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7 January 2021

Candidate proto-oncogene bcl-3 encodes a subunit-specific inhibitor of transcription factor NF-kappa B

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7 January 2021

Cutting edge: a novel Toll/IL-1 receptor domain-containing adapter that preferentially activates the IFN-beta promoter in the Toll-like receptor signaling

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7 January 2021

Role of adaptor TRIF in the MyD88-independent toll-like receptor signaling pathway

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7 January 2021

TIR domain-containing adaptors define the specificity of TLR signaling

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7 January 2021

Regulation of Toll/IL-1-receptor-mediated gene expression by the inducible nuclear protein IkappaBzeta

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7 January 2021

A novel IkappaB protein, IkappaB-zeta, induced by proinflammatory stimuli, negatively regulates nuclear factor-kappaB in the nuclei

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7 January 2021

Stimulus-specific induction of a novel nuclear factor-kappaB regulator, IkappaB-zeta, via Toll/Interleukin-1 receptor is mediated by mRNA stabilization

1 reference

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7 January 2021

10.1016/S0083-6729(06)74012-2

Identifiers

DOI

1 reference

15 August 2017

1 reference