(Q36798371)

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Celecoxib reduces brain dopaminergic neuronaldysfunction, and improves sensorimotor behavioral performance in neonatal rats exposed to systemic lipopolysaccharide.

scientific article published on 5 April 2013

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28 February 2020

Celecoxib reduces brain dopaminergic neuronaldysfunction, and improves sensorimotor behavioral performance in neonatal rats exposed to systemic lipopolysaccharide (English)

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28 February 2020

lipopolysaccharide

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inferred from title

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3

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28 February 2020

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Asuka Kaizaki

1

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28 February 2020

Lu-Tai Tien

2

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28 February 2020

Zhengwei Cai

4

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28 February 2020

Sachiko Tanaka

5

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28 February 2020

Satoshi Numazawa

6

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28 February 2020

Abhay J Bhatt

7

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28 February 2020

Lir-Wan Fan

8

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28 February 2020

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5 April 2013

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28 February 2020

Journal of Neuroinflammation

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28 February 2020

10

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28 February 2020

45

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28 February 2020

copyright license

Creative Commons Attribution 2.0 Generic

5 April 2013

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April 2022 Public Data File from Crossref

copyright status

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April 2022 Public Data File from Crossref

Models of fetal brain injury, intrauterine inflammation, and preterm birth

1 reference

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Induction of COX-2 enzyme and down-regulation of COX-1 expression by lipopolysaccharide (LPS) control prostaglandin E2 production in astrocytes

1 reference

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Neonatal exposure to lipopolysaccharide enhances vulnerability of nigrostriatal dopaminergic neurons to rotenone neurotoxicity in later life

1 reference

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1 reference

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1 reference

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Interleukin-1beta-induced brain injury in the neonatal rat can be ameliorated by alpha-phenyl-n-tert-butyl-nitrone

1 reference

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29 September 2017

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Mechanisms underlying methamphetamine-induced dopamine transporter complex formation

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29 September 2017

Cognitive and cerebral metabolic effects of celecoxib versus placebo in people with age-related memory loss: randomized controlled study

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29 September 2017

Effects of endotoxin derived from Escherichia coli lipopolysaccharide on the pharmacokinetics of drugs

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29 September 2017

alpha-Phenyl-n-tert-butyl-nitrone attenuates lipopolysaccharide-induced brain injury and improves neurological reflexes and early sensorimotor behavioral performance in juvenile rats.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3637465

29 September 2017

Cyclooxygenase-2 is involved in oxidative damage and alpha-synuclein accumulation in dopaminergic cells

1 reference

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29 September 2017

Alpha-Phenyl-n-tert-butyl-nitrone attenuates lipopolysaccharide-induced neuronal injury in the neonatal rat brain

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3637465

29 September 2017

Glial reactions in Parkinson's disease

1 reference

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29 September 2017

The cardioprotection of the late phase of ischemic preconditioning is enhanced by postconditioning via a COX-2-mediated mechanism in conscious rats

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Therapeutic approaches to inflammation in neurodegenerative disease

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Extrapolating brain development from experimental species to humans.

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Relative thromboembolic risks associated with COX-2 inhibitors

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Selective COX-2 inhibition prevents progressive dopamine neuron degeneration in a rat model of Parkinson's disease

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5 September 2018

alpha-Phenyl-n-tert-butyl-nitrone reduces lipopolysaccharide-induced white matter injury in the neonatal rat brain

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3637465

5 September 2018

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Post-training intrahippocampal infusion of nicotine prevents spatial memory retention deficits induced by the cyclo-oxygenase-2-specific inhibitor celecoxib in rats.

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Minocycline attenuates lipopolysaccharide-induced white matter injury in the neonatal rat brain.

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12 December 2020

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10.1186/1742-2094-10-45

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28 February 2020

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28 February 2020

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Europe PubMed Central

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28 February 2020

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