(Q36799163)

16 August 2017

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title

The permeability transition pore in cell death (English)

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16 August 2017

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Stefan Grimm

1

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16 August 2017

Dieter Brdiczka

series ordinal

2

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16 August 2017

language of work or name

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1 May 2007

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16 August 2017

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16 August 2017

volume

12

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16 August 2017

issue

5

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16 August 2017

page(s)

841-855

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A heart mitochondrial Ca2(+)-dependent pore of possible relevance to re-perfusion-induced injury. Evidence that ADP facilitates pore interconversion between the closed and open states

16 August 2017

cites work

1 reference

https://api.crossref.org/works/10.1007%2FS10495-007-0747-3

The mitochondrial permeability transition: its molecular mechanism and role in reperfusion injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10495-007-0747-3

Permeation of hydrophilic solutes through mitochondrial outer membranes: review on mitochondrial porins

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10495-007-0747-3

Intracellular compartmentation, structure and function of creatine kinase isoenzymes in tissues with high and fluctuating energy demands: the 'phosphocreatine circuit' for cellular energy homeostasis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10495-007-0747-3

Mitochondrial contact sites: their role in energy metabolism and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10495-007-0747-3

21 January 2018

Lipids of mitochondria

1 reference

https://api.crossref.org/works/10.1007%2FS10495-007-0747-3

Inhibition of Ca2(+)-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adeni

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10495-007-0747-3

10.1007/S10495-007-0747-3

21 January 2018

Identifiers

DOI

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16 August 2017

1 reference