(Q37138742)

18 August 2017

Redox signaling at reperfusion is required for protection from ischemic preconditioning but not from a direct PKC activator (English)

1 reference

18 August 2017

Yanping Liu

1

1 reference

18 August 2017

Xi-Ming Yang

2

1 reference

18 August 2017

Efstathios K Iliodromitis

3

1 reference

18 August 2017

Dimitrios T Kremastinos

4

1 reference

18 August 2017

Turhan Dost

5

1 reference

18 August 2017

Michael V Cohen

6

1 reference

18 August 2017

James M Downey

7

1 reference

18 August 2017

language of work or name

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12 November 2007

1 reference

Basic Research in Cardiology

18 August 2017

1 reference

18 August 2017

103

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18 August 2017

1

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18 August 2017

54-59

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https://scigraph.springernature.com/pub.10.1007/s00395-007-0683-y

18 August 2017

0 references

cites work

Protein kinase C protects preconditioned rabbit hearts by increasing sensitivity of adenosine A2b-dependent signaling during early reperfusion

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

GSK3beta inhibition and K(ATP) channel opening mediate acute opioid-induced cardioprotection at reperfusion

14 September 2017

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

Cardioprotection and mitochondrial S-nitrosation: effects of S-nitroso-2-mercaptopropionyl glycine (SNO-MPG) in cardiac ischemia-reperfusion injury

14 September 2017

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

Postconditioning protects rabbit hearts through a protein kinase C-adenosine A2b receptor cascade

14 September 2017

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore

14 September 2017

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

Preconditioning protects by inhibiting the mitochondrial permeability transition

14 September 2017

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

Mechanism of myocardial "stunning".

14 September 2017

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

Zinc release from protein kinase C as the common event during activation by lipid second messenger or reactive oxygen

14 September 2017

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

Marked reduction of free radical generation and contractile dysfunction by antioxidant therapy begun at the time of reperfusion. Evidence that myocardial "stunning" is a manifestation of reperfusion injury

29 September 2017

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

Post-conditioning induced cardioprotection requires signaling through a redox-sensitive mechanism, mitochondrial ATP-sensitive K+ channel and protein kinase C activation.

29 September 2017

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

Endogenous adenosine protects preconditioned heart during early minutes of reperfusion by activating Akt.

3 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

ROS and NO trigger early preconditioning: relationship to mitochondrial KATP channel

3 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

Opening of ATP-sensitive potassium channels causes generation of free radicals in vascular smooth muscle cells

3 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

Opening of mitochondrial K(ATP) channels triggers the preconditioned state by generating free radicals.

3 September 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2660167

Oxygen radicals released during ischemic preconditioning contribute to cardioprotection in the rabbit myocardium

3 September 2018

1 reference

12 December 2020

A really radical observation--a comment on Penna et al. in Basic Res Cardiol (2006) 101:180-189

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12 December 2020

Oxygen radicals can induce preconditioning in rabbit hearts

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12 December 2020

Ischemic preconditioning protects by activating prosurvival kinases at reperfusion

1 reference

12 December 2020

The mechanism by which the mitochondrial ATP-sensitive K+ channel opening and H2O2 inhibit the mitochondrial permeability transition

1 reference

12 December 2020

Opening mitoKATP increases superoxide generation from complex I of the electron transport chain

1 reference

12 December 2020

The pH hypothesis of postconditioning: staccato reperfusion reintroduces oxygen and perpetuates myocardial acidosis

1 reference

12 December 2020

Ischemic preconditioning targets the reperfusion phase

1 reference

12 December 2020

Preconditioning in cardiomyocytes protects by attenuating oxidant stress at reperfusion

1 reference

12 December 2020

Evidence that translocation of protein kinase C is a key event during ischemic preconditioning of rabbit myocardium

1 reference

12 December 2020

10.1007/S00395-007-0683-Y

Identifiers

DOI

1 reference

18 August 2017

1 reference

18 August 2017

PubMed publication ID

17999029

1 reference