(Q37149749)

English

The formation of double-strand breaks at multiply damaged sites is driven by the kinetics of excision/incision at base damage in eukaryotic cells

scientific article published on 27 January 2009

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

The formation of double-strand breaks at multiply damaged sites is driven by the kinetics of excision/incision at base damage in eukaryotic cells (English)

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

author name string

Stanislav G Kozmin

1

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

Yuliya Sedletska

2

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

Anne Reynaud-Angelin

3

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

Didier Gasparutto

4

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

Evelyne Sage

5

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

language of work or name

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publication date

27 January 2009

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

Nucleic Acids Research

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Europe PubMed Central

PMC publication ID

18 August 2017

37

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

6

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

1767-1777

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

describes a project that uses

1 reference

Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC2665211/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

DNA repair of clustered lesions in mammalian cells: involvement of non-homologous end-joining

1 reference

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Closely opposed apurinic/apyrimidinic sites are converted to double strand breaks in Escherichia coli even in the absence of exonuclease III, endonuclease IV, nucleotide excision repair and AP lyase cleavage

1 reference

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1 reference

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Efficiency of incision of an AP site within clustered DNA damage by the major human AP endonuclease

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Base excision repair processing of radiation-induced clustered DNA lesions.

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Reactivity of human apurinic/apyrimidinic endonuclease and Escherichia coli exonuclease III with bistranded abasic sites in DNA.

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Saccharomyces cerevisiae Ku70 potentiates illegitimate DNA double-strand break repair and serves as a barrier to error-prone DNA repair pathways

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Cell cycle and genetic requirements of two pathways of nonhomologous end-joining repair of double-strand breaks in Saccharomyces cerevisiae

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The roles of specific glycosylases in determining the mutagenic consequences of clustered DNA base damage

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1 reference

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In vitro repair of synthetic ionizing radiation-induced multiply damaged DNA sites

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Catalytic and DNA binding properties of the ogg1 protein of Saccharomyces cerevisiae: comparison between the wild type and the K241R and K241Q active-site mutant proteins

1 reference

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Identifiers

10.1093/NAR/GKP010

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

ResearchGate publication ID

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