(Q37384718)

20 August 2017

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title

Latest findings in phosphate homeostasis (English)

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20 August 2017

Dominique Prié

1

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20 August 2017

Pablo Ureña Torres

series ordinal

2

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20 August 2017

Gérard Friedlander

series ordinal

3

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20 August 2017

language of work or name

English

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publication date

4 February 2009

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20 August 2017

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20 August 2017

75

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20 August 2017

9

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20 August 2017

882-889

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https://scigraph.springernature.com/pub.10.1038/ki.2008.643

20 August 2017

0 references

cites work

Recent findings in phosphate homeostasis

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Association of serum phosphorus and calcium x phosphate product with mortality risk in chronic hemodialysis patients: a national study

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Relation between serum phosphate level and cardiovascular event rate in people with coronary disease

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Sodium-phosphate cotransporters, nephrolithiasis and bone demineralization.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Proximal tubular phosphate reabsorption: molecular mechanisms

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Cloning, gene structure and dietary regulation of the type-IIc Na/Pi cotransporter in the mouse kidney

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Targeted inactivation of Npt2 in mice leads to severe renal phosphate wasting, hypercalciuria, and skeletal abnormalities

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Nephrolithiasis and osteoporosis associated with hypophosphatemia caused by mutations in the type 2a sodium-phosphate cotransporter

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Renal calcification in mice homozygous for the disrupted type IIa Na/Pi cotransporter gene Npt2.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

NPT2a gene variation in calcium nephrolithiasis with renal phosphate leak.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

SLC34A3 mutations in patients with hereditary hypophosphatemic rickets with hypercalciuria predict a key role for the sodium-phosphate cotransporter NaPi-IIc in maintaining phosphate homeostasis

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Novel regulators of phosphate homeostasis and bone metabolism

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Hereditary hypophosphatemic rickets with hypercalciuria is caused by mutations in the sodium-phosphate cotransporter gene SLC34A3.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Intronic deletions in the SLC34A3 gene cause hereditary hypophosphatemic rickets with hypercalciuria

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Renouncing electroneutrality is not free of charge: switching on electrogenicity in a Na+-coupled phosphate cotransporter.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Cloning and functional characterization of a sodium-dependent phosphate transporter expressed in human lung and small intestine

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Molecular cloning, functional characterization, tissue distribution, and chromosomal localization of a human, small intestinal sodium-phosphate (Na+-Pi) transporter (SLC34A2)

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Regulation of intestinal Na+-dependent phosphate co-transporters by a low-phosphate diet and 1,25-dihydroxyvitamin D3

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Mutations in SLC34A2 cause pulmonary alveolar microlithiasis and are possibly associated with testicular microlithiasis

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Localization of NaPi-1, a Na/Pi cotransporter, in rabbit kidney proximal tubules. II. Localization by immunohistochemistry

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Expression of a renal type I sodium/phosphate transporter (NaPi-1) induces a conductance in Xenopus oocytes permeable for organic and inorganic anions

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

The SLC20 family of proteins: dual functions as sodium-phosphate cotransporters and viral receptors

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Transmembrane topology of PiT-2, a phosphate transporter-retrovirus receptor

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Vascular calcification mechanisms

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Proximal tubular handling of phosphate: A molecular perspective

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Npt2 gene disruption confers resistance to the inhibitory action of parathyroid hormone on renal sodium-phosphate cotransport

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Genetic evidence of serum phosphate-independent functions of FGF-23 on bone

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Interaction of the type IIa Na/Pi cotransporter with PDZ proteins

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Na(+)/H(+ ) exchanger regulatory factor 2 directs parathyroid hormone 1 receptor signalling

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Targeted disruption of the mouse NHERF-1 gene promotes internalization of proximal tubule sodium-phosphate cotransporter type IIa and renal phosphate wasting

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Defective coupling of apical PTH receptors to phospholipase C prevents internalization of the Na+-phosphate cotransporter NaPi-IIa in Nherf1-deficient mice

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Na+/H+ exchanger-regulatory factor 1 mediates inhibition of phosphate transport by parathyroid hormone and second messengers by acting at multiple sites in opossum kidney cells

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Truncation of the carboxyl-terminal region of the rat parathyroid hormone (PTH)/PTH-related peptide receptor enhances PTH stimulation of adenylyl cyclase but not phospholipase C.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

NHERF1 mutations and responsiveness of renal parathyroid hormone

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Bone as a source of FGF23: regulation by phosphate?

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Pathogenic role of Fgf23 in Hyp mice.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Homozygous ablation of fibroblast growth factor-23 results in hyperphosphatemia and impaired skeletogenesis, and reverses hypophosphatemia in Phex-deficient mice

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Fibroblast growth factor-23 relationship to dietary phosphate and renal phosphate handling in healthy young men.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

FGF-23 is elevated by chronic hyperphosphatemia

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

1alpha,25-Dihydroxyvitamin D3 upregulates FGF23 gene expression in bone: the final link in a renal-gastrointestinal-skeletal axis that controls phosphate transport

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Fibroblast growth factor 23 is a counter-regulatory phosphaturic hormone for vitamin D.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Regulation of C-terminal and intact FGF-23 by dietary phosphate in men and women

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Circulating FGF-23 is regulated by 1alpha,25-dihydroxyvitamin D3 and phosphorus in vivo

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Dietary phosphorus regulates serum fibroblast growth factor-23 concentrations in healthy men.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Autosomal-dominant hypophosphatemic rickets (ADHR) mutations stabilize FGF-23

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

The autosomal dominant hypophosphatemic rickets (ADHR) gene is a secreted polypeptide overexpressed by tumors that cause phosphate wasting

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Transgenic mice expressing fibroblast growth factor 23 under the control of the alpha1(I) collagen promoter exhibit growth retardation, osteomalacia, and disturbed phosphate homeostasis

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Increased circulatory level of biologically active full-length FGF-23 in patients with hypophosphatemic rickets/osteomalacia

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Human fibroblast growth factor-23 mutants suppress Na+-dependent phosphate co-transport activity and 1alpha,25-dihydroxyvitamin D3 production

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

FGF-23 is a potent regulator of vitamin D metabolism and phosphate homeostasis

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

FGF-23 transgenic mice demonstrate hypophosphatemic rickets with reduced expression of sodium phosphate cotransporter type IIa.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Role of the vitamin D receptor in FGF23 action on phosphate metabolism

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Fibroblast growth factor-23 regulates parathyroid hormone and 1alpha-hydroxylase expression in cultured bovine parathyroid cells

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Premature aging-like phenotype in fibroblast growth factor 23 null mice is a vitamin D-mediated process

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Genetic ablation of vitamin D activation pathway reverses biochemical and skeletal anomalies in Fgf-23-null animals

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Anti-FGF23 neutralizing antibodies show the physiological role and structural features of FGF23.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

A novel GALNT3 mutation in a pseudoautosomal dominant form of tumoral calcinosis: evidence that the disorder is autosomal recessive

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

A novel recessive mutation in fibroblast growth factor-23 causes familial tumoral calcinosis

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

The role of mutant UDP-N-acetyl-alpha-D-galactosamine-polypeptide N-acetylgalactosaminyltransferase 3 in regulating serum intact fibroblast growth factor 23 and matrix extracellular phosphoglycoprotein in heritable tumoral calcinosis.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Hyperostosis-hyperphosphatemia syndrome: a congenital disorder of O-glycosylation associated with augmented processing of fibroblast growth factor 23.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

A novel mutation in fibroblast growth factor 23 gene as a cause of tumoral calcinosis

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

A novel homozygous missense mutation in FGF23 causes Familial Tumoral Calcinosis associated with disseminated visceral calcification

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Ablation of vitamin D signaling rescues bone, mineral, and glucose homeostasis in Fgf-23 deficient mice.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Role of hyperphosphatemia and 1,25-dihydroxyvitamin D in vascular calcification and mortality in fibroblastic growth factor 23 null mice

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Mutation of the mouse klotho gene leads to a syndrome resembling ageing

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Klotho, a gene related to a syndrome resembling human premature aging, functions in a negative regulatory circuit of vitamin D endocrine system

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Regulation of fibroblast growth factor-23 signaling by klotho

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Klotho converts canonical FGF receptor into a specific receptor for FGF23

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Immunohistochemical localization of Klotho protein in brain, kidney, and reproductive organs of mice.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Klotho is a novel beta-glucuronidase capable of hydrolyzing steroid beta-glucuronides

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

The beta-glucuronidase klotho hydrolyzes and activates the TRPV5 channel

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Suppression of aging in mice by the hormone Klotho

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Regulation of oxidative stress by the anti-aging hormone klotho

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Fibroblast growth factor-23 mitigates hyperphosphatemia but accentuates calcitriol deficiency in chronic kidney disease

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Circulating concentration of FGF-23 increases as renal function declines in patients with chronic kidney disease, but does not change in response to variation in phosphate intake in healthy volunteers

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Serum FGF23 levels in normal and disordered phosphorus homeostasis.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Bone mass does not correlate with the serum fibroblast growth factor 23 in hemodialysis patients

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Fibroblast Growth Factor 23 (FGF23) Predicts Progression of Chronic Kidney Disease: The Mild to Moderate Kidney Disease (MMKD) Study

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Pretreatment serum FGF-23 levels predict the efficacy of calcitriol therapy in dialysis patients.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Serum fibroblast growth factor-23 levels predict the future refractory hyperparathyroidism in dialysis patients.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Post-transplant hypophosphatemia: Tertiary 'Hyper-Phosphatoninism'?

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Severely reduced production of klotho in human chronic renal failure kidney.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Association between a functional variant of the KLOTHO gene and high-density lipoprotein cholesterol, blood pressure, stroke, and longevity

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

KLOTHO allele status and the risk of early-onset occult coronary artery disease

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Association of human aging with a functional variant of klotho

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

The differential effects of age on the association of KLOTHO gene polymorphisms with coronary artery disease.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Association of the F352V variant of the Klotho gene with bone mineral density

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Association of klotho gene polymorphism with bone density and spondylosis of the lumbar spine in postmenopausal women

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Klotho gene polymorphisms associated with bone density of aged postmenopausal women

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Klotho expression in epithelial ovarian cancer and its association with insulin-like growth factors and disease progression

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Anti-apoptotic and anti-senescence effects of Klotho on vascular endothelial cells

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Klotho reduces apoptosis in experimental ischaemic acute renal failure

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Regulation of angiogenesis by the aging suppressor gene klotho

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Angiogenesis and vasculogenesis are impaired in the precocious-aging klotho mouse

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Matrix extracellular phosphoglycoprotein causes phosphaturia in rats by inhibiting tubular phosphate reabsorption

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

MEPE has the properties of an osteoblastic phosphatonin and minhibin.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

MEPE, a new gene expressed in bone marrow and tumors causing osteomalacia

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Mepe, the gene encoding a tumor-secreted protein in oncogenic hypophosphatemic osteomalacia, is expressed in bone

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Serum MEPE-ASARM-peptides are elevated in X-linked rickets (HYP): implications for phosphaturia and rickets

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Regulation of fibroblastic growth factor 23 expression but not degradation by PHEX.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Phosphorylated acidic serine-aspartate-rich MEPE-associated motif peptide from matrix extracellular phosphoglycoprotein inhibits phosphate regulating gene with homologies to endopeptidases on the X-chromosome enzyme activity

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Degradation of MEPE, DMP1, and release of SIBLING ASARM-peptides (minhibins): ASARM-peptide(s) are directly responsible for defective mineralization in HYP

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Surface plasmon resonance (SPR) confirms that MEPE binds to PHEX via the MEPE-ASARM motif: a model for impaired mineralization in X-linked rickets (HYP)

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Role of matrix extracellular phosphoglycoprotein in the pathogenesis of X-linked hypophosphatemia

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolism.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

DMP1 mutations in autosomal recessive hypophosphatemia implicate a bone matrix protein in the regulation of phosphate homeostasis.

1 reference

https://api.crossref.org/works/10.1038%2FKI.2008.643

7 January 2021

Identifiers

DOI

10.1038/KI.2008.643

1 reference

Dimensions Publication ID

20 August 2017

0 references

1 reference