(Q37442573)

English

The nucleus tractus solitarius: a portal for visceral afferent signal processing, energy status assessment and integration of their combined effects on food intake

scientific article published on April 2009

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

20 August 2017

1 reference

Europe PubMed Central

The nucleus tractus solitarius: a portal for visceral afferent signal processing, energy status assessment and integration of their combined effects on food intake (English)

1 reference

Europe PubMed Central

PubMed publication ID

20 August 2017

signal processing

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author name string

H J Grill

1

1 reference

Europe PubMed Central

PubMed publication ID

20 August 2017

M R Hayes

2

1 reference

Europe PubMed Central

PubMed publication ID

20 August 2017

language of work or name

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publication date

1 April 2009

1 reference

Europe PubMed Central

PubMed publication ID

20 August 2017

International Journal of Obesity

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Europe PubMed Central

PubMed publication ID

20 August 2017

33 Suppl 1

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Europe PubMed Central

PubMed publication ID

20 August 2017

S11-5

1 reference

Europe PubMed Central

PubMed publication ID

20 August 2017

https://scigraph.springernature.com/pub.10.1038/ijo.2009.10

0 references

The direct and indirect controls of meal size

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Peripheral exendin-4 and peptide YY(3-36) synergistically reduce food intake through different mechanisms in mice

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The inhibitory effects of peripheral administration of peptide YY(3-36) and glucagon-like peptide-1 on food intake are attenuated by ablation of the vagal-brainstem-hypothalamic pathway

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Intravenous infusion of glucagon-like peptide-1 potently inhibits food intake, sham feeding, and gastric emptying in rats.

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Glucagon-like peptide-1 7-36: a physiological incretin in man.

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Energy expenditure by intravenous administration of glucagon-like peptide-1 mediated by the lower brainstem and sympathoadrenal system

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Distribution of glucagon-like peptide-1 and other preproglucagon-derived peptides in the rat hypothalamus and brainstem.

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Antiobesity action of peripheral exenatide (exendin-4) in rodents: effects on food intake, body weight, metabolic status and side-effect measures

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Projections from the nucleus of the solitary tract in the rat

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Ascending central gustatory pathways

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Peptides that regulate food intake: glucagon-like peptide 1-(7-36) amide acts at lateral and medial hypothalamic sites to suppress feeding in rats

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Differential hypothalamic neuronal activation following peripheral injection of GLP-1 and oxyntomodulin in mice detected by manganese-enhanced magnetic resonance imaging.

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Glucagon-like peptide 1 stimulates hypothalamic proopiomelanocortin neurons

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Exendin-4 treatment improves metabolic control after rat islet transplantation to athymic mice with streptozotocin-induced diabetes.

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Exenatide: a GLP-1 receptor agonist as novel therapy for Type 2 diabetes mellitus

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Exenatide: a new option for the treatment of type 2 diabetes

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Effect of occluding the pylorus on intraoral intake: a test of the gastric hypothesis of meal termination

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5-HT3 receptors participate in CCK-induced suppression of food intake by delaying gastric emptying

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Oxyntomodulin and glucagon-like peptide-1 differentially regulate murine food intake and energy expenditure

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The neuroanatomical axis for control of energy balance

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Decreased intake of a liquid diet in nonfood-deprived rats following intra-PVN injections of GLP-1 (7-36) amide.

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Characterization of brainstem preproglucagon projections to the paraventricular and dorsomedial hypothalamic nuclei

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Leptin regulation of neuroendocrine systems

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Central nervous system control of food intake

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Local gene knockdown in the brain using viral-mediated RNA interference.

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Leptin receptor signaling in midbrain dopamine neurons regulates feeding

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Leptin regulation of the mesoaccumbens dopamine pathway.

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Synergy between leptin and cholecystokinin (CCK) to control daily caloric intake

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Leptin and the control of food intake: neurons in the nucleus of the solitary tract are activated by both gastric distension and leptin

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Leptin and neuropeptide y have opposing modulatory effects on nucleus of the solitary tract neurophysiological responses to gastric loads: implications for the control of food intake.

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Gastric distension enhances CCK-induced Fos-like immunoreactivity in the dorsal hindbrain by activating 5-HT3 receptors.

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Distribution and structure of vagal afferent intraganglionic laminar endings (IGLEs) in the rat gastrointestinal tract

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Gastric distension induces c-Fos in medullary GLP-1/2-containing neurons

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AMP-kinase regulates food intake by responding to hormonal and nutrient signals in the hypothalamus

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AMPK integrates nutrient and hormonal signals to regulate food intake and energy balance through effects in the hypothalamus and peripheral tissues

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Glucagon-like peptide-1: a potent regulator of food intake in humans

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Identifiers

10.1038/IJO.2009.10

1 reference

Europe PubMed Central

PubMed publication ID

20 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

20 August 2017

ResearchGate publication ID

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