(Q37861557)

24 August 2017

title

The secreted protease factor CPAF is responsible for degrading pro-apoptotic BH3-only proteins in Chlamydia trachomatis-infected cells (English)

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24 August 2017

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Chlamydia trachomatis

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author name string

Mustak Pirbhai

1

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24 August 2017

Feng Dong

2

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24 August 2017

Youmin Zhong

3

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24 August 2017

Kelvin Z Pan

4

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24 August 2017

Guangming Zhong

5

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24 August 2017

language of work or name

English

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publication date

28 August 2006

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Journal of Biological Chemistry

24 August 2017

number of pages

7

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based on heuristic

inferred from page(s)

published in

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24 August 2017

volume

281

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24 August 2017

issue

42

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24 August 2017

page(s)

31495-31501

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Secretion of proinflammatory cytokines by epithelial cells in response to Chlamydia infection suggests a central role for epithelial cells in chlamydial pathogenesis

24 August 2017

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Inhibition of apoptosis in chlamydia-infected cells: blockade of mitochondrial cytochrome c release and caspase activation

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Chlamydia inhibits interferon gamma-inducible major histocompatibility complex class II expression by degradation of upstream stimulatory factor 1.

21 January 2018

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Degradation of transcription factor RFX5 during the inhibition of both constitutive and interferon gamma-inducible major histocompatibility complex class I expression in chlamydia-infected cells

21 January 2018

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Identification of a chlamydial protease-like activity factor responsible for the degradation of host transcription factors

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Chlamydia-infected cells continue to undergo mitosis and resist induction of apoptosis

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Cleavage of host keratin 8 by a Chlamydia-secreted protease

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Intramolecular dimerization is required for the chlamydia-secreted protease CPAF to degrade host transcriptional factors

21 January 2018

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Cleavage-dependent activation of a chlamydia-secreted protease

21 January 2018

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Persistent Chlamydia trachomatis infections resist apoptotic stimuli

21 January 2018

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

21 January 2018

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Cell damage-induced conformational changes of the pro-apoptotic protein Bak in vivo precede the onset of apoptosis

21 January 2018

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Bax-induced cytochrome c release from mitochondria depends on alpha-helices-5 and -6

21 January 2018

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Bax and Bak coalesce into novel mitochondria-associated clusters during apoptosis

21 January 2018

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21 January 2018

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21 January 2018

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Chlamydia trachomatis infection inhibits both Bax and Bak activation induced by staurosporine

21 January 2018

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Degradation of the proapoptotic proteins Bik, Puma, and Bim with Bcl-2 domain 3 homology in Chlamydia trachomatis-infected cells

21 January 2018

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Broad degradation of proapoptotic proteins with the conserved Bcl-2 homology domain 3 during infection with Chlamydia trachomatis

21 January 2018

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Chlamydia inhibit host cell apoptosis by degradation of proapoptotic BH3-only proteins

21 January 2018

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NF-kappa B activation is not required for Chlamydia trachomatis inhibition of host epithelial cell apoptosis

21 January 2018

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Production, specificity, and functionality of monoclonal antibodies to specific peptide–major histocompatibility complex class II complexes formed by processing of exogenous protein

21 January 2018

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Evidence that binding site occupancy is necessary and sufficient for effective major histocompatibility complex (MHC) class II transport through the secretory pathway redefines the primary function of class II-associated invariant chain peptides (CL

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M602796200

21 January 2018

Identifiers

DOI

10.1074/JBC.M602796200

1 reference

24 August 2017

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