(Q37917873)

25 August 2017

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title

Nitric oxide synthase and cyclic GMP signaling in cardiac myocytes: from contractility to remodeling (English)

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25 August 2017

Joanna Hammond

1

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25 August 2017

Jean-Luc Balligand

series ordinal

2

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25 August 2017

language of work or name

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7 August 2011

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Journal of Molecular and Cellular Cardiology

25 August 2017

published in

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25 August 2017

volume

52

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25 August 2017

issue

2

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25 August 2017

page(s)

330-340

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A rapid and potent natriuretic response to intravenous injection of atrial myocardial extract in rats

25 August 2017

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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PDE5 inhibition with sildenafil improves left ventricular diastolic function, cardiac geometry, and clinical status in patients with stable systolic heart failure: results of a 1-year, prospective, randomized, placebo-controlled study.

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Nitric oxide, atrial natriuretic peptide, and cyclic GMP inhibit the growth-promoting effects of norepinephrine in cardiac myocytes and fibroblasts

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Endothelial nitric oxide synthase limits left ventricular remodeling after myocardial infarction in mice.

7 January 2021

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Cardiomyocyte-restricted restoration of nitric oxide synthase 3 attenuates left ventricular remodeling after chronic pressure overload

7 January 2021

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Pressure overload-induced LV hypertrophy and dysfunction in mice are exacerbated by congenital NOS3 deficiency.

7 January 2021

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7 January 2021

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7 January 2021

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nNOS gene deletion exacerbates pathological left ventricular remodeling and functional deterioration after myocardial infarction

7 January 2021

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Deficiency of neuronal nitric oxide synthase increases mortality and cardiac remodeling after myocardial infarction: role of nitroso-redox equilibrium

7 January 2021

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Conditional neuronal nitric oxide synthase overexpression impairs myocardial contractility

7 January 2021

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7 January 2021

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Regulation of myocyte contraction via neuronal nitric oxide synthase: role of ryanodine receptor S-nitrosylation

7 January 2021

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Reduced phospholamban phosphorylation is associated with impaired relaxation in left ventricular myocytes from neuronal NO synthase-deficient mice.

7 January 2021

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The fork in the nitric oxide road: cyclic GMP or nitrosylation?

7 January 2021

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Nitric oxide synthase 2 and pressure-overload-induced left ventricular remodelling in mice.

7 January 2021

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7 January 2021

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7 January 2021

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Cardiomyocyte overexpression of iNOS in mice results in peroxynitrite generation, heart block, and sudden death

7 January 2021

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Cardiac-specific overexpression of inducible nitric oxide synthase does not result in severe cardiac dysfunction

7 January 2021

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Acute inhibition of myoglobin impairs contractility and energy state of iNOS-overexpressing hearts

7 January 2021

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7 January 2021

The Janus faces of NO?

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7 January 2021

The Janus Faces of iNOS

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Sildenafil citrate increases myocardial cGMP content in rat heart, decreases its hypertrophic response to isoproterenol and decreases myocardial leak of creatine kinase and troponin T

7 January 2021

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Role of cyclic GMP-dependent protein kinase in the contractile response to exogenous nitric oxide in rat cardiac myocytes

7 January 2021

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Gene transfer of cGMP-dependent protein kinase I enhances the antihypertrophic effects of nitric oxide in cardiomyocytes.

7 January 2021

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Defective smooth muscle regulation in cGMP kinase I-deficient mice

7 January 2021

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Rescue of cGMP kinase I knockout mice by smooth muscle specific expression of either isozyme

7 January 2021

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Regulation and role of myocyte cyclic GMP-dependent protein kinase-1

7 January 2021

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A calcineurin-dependent transcriptional pathway for cardiac hypertrophy

7 January 2021

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Targeted inhibition of calcineurin attenuates cardiac hypertrophy in vivo

7 January 2021

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Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy

7 January 2021

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The delta isoform of CaM kinase II is required for pathological cardiac hypertrophy and remodeling after pressure overload

7 January 2021

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Requirement for Ca2+/calmodulin-dependent kinase II in the transition from pressure overload-induced cardiac hypertrophy to heart failure in mice.

7 January 2021

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Inhibition of calcineurin-NFAT hypertrophy signaling by cGMP-dependent protein kinase type I in cardiac myocytes

7 January 2021

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Enhanced activity of the myocardial Na+/H+ exchanger NHE-1 contributes to cardiac remodeling in atrial natriuretic peptide receptor-deficient mice.

7 January 2021

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Calcineurin-nuclear factor of activated T cells pathway-dependent cardiac remodeling in mice deficient in guanylyl cyclase A, a receptor for atrial and brain natriuretic peptides

7 January 2021

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Impaired cardiac hypertrophic response in Calcineurin Abeta -deficient mice

7 January 2021

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Phosphodiesterase 5 inhibition blocks pressure overload-induced cardiac hypertrophy independent of the calcineurin pathway

7 January 2021

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Ventricular arrhythmias, increased cardiac calmodulin kinase II expression, and altered repolarization kinetics in ANP receptor deficient mice

7 January 2021

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Single L-type Ca(2+) channel regulation by cGMP-dependent protein kinase type I in adult cardiomyocytes from PKG I transgenic mice

7 January 2021

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Protein kinase G phosphorylates Cav1.2 alpha1c and beta2 subunits

7 January 2021

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Does contractile Ca2+ control calcineurin-NFAT signaling and pathological hypertrophy in cardiac myocytes?

7 January 2021

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Phosphodiesterase 5A inhibition induces Na+/H+ exchanger blockade and protection against myocardial infarction

7 January 2021

1 reference

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Decreased activity of the Na+/H+ exchanger by phosphodiesterase 5A inhibition is attributed to an increase in protein phosphatase activity

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

Activation of Na+/H+ exchanger 1 is sufficient to generate Ca2+ signals that induce cardiac hypertrophy and heart failure

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

TRPC channels as effectors of cardiac hypertrophy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

Canonical transient receptor potential channels promote cardiomyocyte hypertrophy through activation of calcineurin signaling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

TRPC6 fulfills a calcineurin signaling circuit during pathologic cardiac remodeling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

TRPC3 and TRPC6 are essential for angiotensin II-induced cardiac hypertrophy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

TRPC channels are necessary mediators of pathologic cardiac hypertrophy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

Cyclic GMP/PKG-dependent inhibition of TRPC6 channel activity and expression negatively regulates cardiomyocyte NFAT activation Novel mechanism of cardiac stress modulation by PDE5 inhibition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

Inhibition of TRPC6 channel activity contributes to the antihypertrophic effects of natriuretic peptides-guanylyl cyclase-A signaling in the heart.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

Phosphorylation of TRPC6 channels at Thr69 is required for anti-hypertrophic effects of phosphodiesterase 5 inhibition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

Regulator of G-protein signaling subtype 4 mediates antihypertrophic effect of locally secreted natriuretic peptides in the heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

Phenotypic spectrum caused by transgenic overexpression of activated Akt in the heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

Distinct roles of GSK-3alpha and GSK-3beta phosphorylation in the heart under pressure overload

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

Cyclic GMP-dependent protein kinase Ialpha attenuates necrosis and apoptosis following ischemia/reoxygenation in adult cardiomyocyte

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

Protein kinase G-dependent cardioprotective mechanism of phosphodiesterase-5 inhibition involves phosphorylation of ERK and GSK3beta

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2011.07.029

10.1016/J.YJMCC.2011.07.029

7 January 2021

Identifiers

DOI

1 reference

25 August 2017

1 reference