(Q37958977)

25 August 2017

title

Defective DNA-damage repair induced by nuclear lamina dysfunction is a key mediator of smooth muscle cell aging (English)

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25 August 2017

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Derek T Warren

1

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25 August 2017

Catherine M Shanahan

series ordinal

2

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25 August 2017

publication date

1 December 2011

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Biochemical Society Transactions

25 August 2017

published in

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25 August 2017

volume

39

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25 August 2017

issue

6

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25 August 2017

page(s)

1780-1785

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Vascular smooth muscle cells undergo telomere-based senescence in human atherosclerosis: effects of telomerase and oxidative stress.

25 August 2017

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Prelamin A acts to accelerate smooth muscle cell senescence and is a novel biomarker of human vascular aging

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DNA damage and repair in atherosclerosis

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Oxidative stress, endothelial dysfunction and atherosclerosis.

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Role of oxidatively induced DNA lesions in human pathogenesis

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The MRN complex: coordinating and mediating the response to broken chromosomes

21 January 2018

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Dial 9-1-1 for DNA damage: the Rad9-Hus1-Rad1 (9-1-1) clamp complex

21 January 2018

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ATM and ATR: components of an integrated circuit

21 January 2018

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The ATM-Chk2 and ATR-Chk1 pathways in DNA damage signaling and cancer

21 January 2018

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PML nuclear bodies: dynamic sensors of DNA damage and cellular stress

21 January 2018

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Evidence for the receipt of DNA damage stimuli by PML nuclear domains

21 January 2018

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Promyelocytic leukemia nuclear bodies behave as DNA damage sensors whose response to DNA double-strand breaks is regulated by NBS1 and the kinases ATM, Chk2, and ATR.

21 January 2018

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Recruitment of NBS1 into PML oncogenic domains via interaction with SP100 protein

21 January 2018

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PML colocalizes with and stabilizes the DNA damage response protein TopBP1

21 January 2018

1 reference

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NBS1 and TRF1 colocalize at promyelocytic leukemia bodies during late S/G2 phases in immortalized telomerase-negative cells. Implication of NBS1 in alternative lengthening of telomeres

21 January 2018

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A nucleolar targeting signal in PML-I addresses PML to nucleolar caps in stressed or senescent cells

21 January 2018

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PML protein association with specific nucleolar structures differs in normal, tumor and senescent human cells

21 January 2018

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Dynamic sorting of nuclear components into distinct nucleolar caps during transcriptional inhibition

21 January 2018

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PML regulates p53 stability by sequestering Mdm2 to the nucleolus

21 January 2018

1 reference

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Physical and functional interactions between PML and MDM2

21 January 2018

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Role of A-type lamins in signaling, transcription, and chromatin organization.

21 January 2018

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The nuclear lamina comes of age.

21 January 2018

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Nesprin-1 and -2 are involved in the pathogenesis of Emery Dreifuss muscular dystrophy and are critical for nuclear envelope integrity

21 January 2018

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The nuclear envelopathies and human diseases

21 January 2018

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Lamin A/C speckles mediate spatial organization of splicing factor compartments and RNA polymerase II transcription

21 January 2018

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Accelerated telomere shortening and replicative senescence in human fibroblasts overexpressing mutant and wild-type lamin A.

21 January 2018

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Genomic instability in laminopathy-based premature aging

21 January 2018

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Nuclear lamins and chromatin: when structure meets function.

21 January 2018

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A-type lamins regulate retinoblastoma protein function by promoting subnuclear localization and preventing proteasomal degradation

21 January 2018

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Novel nuclear nesprin-2 variants tether active extracellular signal-regulated MAPK1 and MAPK2 at promyelocytic leukemia protein nuclear bodies and act to regulate smooth muscle cell proliferation.

21 January 2018

1 reference

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Nesprin-2 is a multi-isomeric protein that binds lamin and emerin at the nuclear envelope and forms a subcellular network in skeletal muscle

21 January 2018

1 reference

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Fast regulation of AP-1 activity through interaction of lamin A/C, ERK1/2, and c-Fos at the nuclear envelope

21 January 2018

1 reference

https://api.crossref.org/works/10.1042%2FBST20110703

Proteasome-dependent dispersal of PML nuclear bodies in response to alkylating DNA damage

21 January 2018

1 reference

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ERK activity facilitates activation of the S-phase DNA damage checkpoint by modulating ATR function

21 January 2018

1 reference

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Extracellular signal-related kinase positively regulates ataxia telangiectasia mutated, homologous recombination repair, and the DNA damage response

21 January 2018

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https://api.crossref.org/works/10.1042%2FBST20110703

Emerin and the nuclear lamina in muscle and cardiac disease.

21 January 2018

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A-type nuclear lamins, progerias and other degenerative disorders.

21 January 2018

1 reference

https://api.crossref.org/works/10.1042%2FBST20110703

Hutchinson-Gilford progeria mutant lamin A primarily targets human vascular cells as detected by an anti-Lamin A G608G antibody

21 January 2018

1 reference

https://api.crossref.org/works/10.1042%2FBST20110703

Mammalian SUN protein interaction networks at the inner nuclear membrane and their role in laminopathy disease processes

21 January 2018

1 reference

https://api.crossref.org/works/10.1042%2FBST20110703

Nesprins LINC the nucleus and cytoskeleton

21 January 2018

1 reference

https://api.crossref.org/works/10.1042%2FBST20110703

Heterochromatin revisited

21 January 2018

1 reference

https://api.crossref.org/works/10.1042%2FBST20110703

Lamin A rod domain mutants target heterochromatin protein 1alpha and beta for proteasomal degradation by activation of F-box protein, FBXW10

21 January 2018

1 reference

https://api.crossref.org/works/10.1042%2FBST20110703

Pre-Lamin A processing is linked to heterochromatin organization.

21 January 2018

1 reference

https://api.crossref.org/works/10.1042%2FBST20110703

Recapitulation of premature ageing with iPSCs from Hutchinson-Gilford progeria syndrome

21 January 2018

1 reference

https://api.crossref.org/works/10.1042%2FBST20110703

21 January 2018

Identifiers

DOI

10.1042/BST20110703

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25 August 2017

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