(Q37980604)

26 August 2017

Tumor necrosis factor-like weak inducer of apoptosis and its potential roles in lupus nephritis (English)

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26 August 2017

Zhi-Chun Liu

1

1 reference

PubMed ID

22297307

26 August 2017

Qiao-Ling Zhou

series ordinal

2

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PubMed ID

22297307

26 August 2017

language of work or name

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1 April 2012

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26 August 2017

Inflammation Research

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26 August 2017

61

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26 August 2017

4

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26 August 2017

277-284

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https://scigraph.springernature.com/pub.10.1007/s00011-011-0420-8

26 August 2017

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cites work

TWEAK, a new secreted ligand in the tumor necrosis factor family that weakly induces apoptosis

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Anti-TWEAK monoclonal antibodies reduce immune cell infiltration in the central nervous system and severity of experimental autoimmune encephalomyelitis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

The role of TWEAK/Fn14 in the pathogenesis of inflammation and systemic autoimmunity

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Tumour necrosis factor-like weak inducer of apoptosis (TWEAK), an important mediator of endothelial inflammation, is associated with the pathogenesis of Henoch-Schonlein purpura

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

No end in site: TWEAK/Fn14 activation and autoimmunity associated- end-organ pathologies

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

TWEAK, a member of the TNF superfamily, is a multifunctional cytokine that binds the TweakR/Fn14 receptor

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

The TWEAK-Fn14 cytokine-receptor axis: discovery, biology and therapeutic targeting

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

The apoptotic ligands TRAIL, TWEAK, and Fas ligand mediate monocyte death induced by autologous lupus T cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Studies on the interaction between TWEAK and the death receptor WSL-1/TRAMP (DR3)

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Interactions of tumor necrosis factor (TNF) and TNF receptor family members in the mouse and human

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Death-inducing tumour necrosis factor (TNF) superfamily ligands and receptors are transcribed in human placentae, cytotrophoblasts, placental macrophages and placental cell lines

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Identification of a ligand for the death-domain-containing receptor Apo3

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

An endogenous hybrid mRNA encodes TWE-PRIL, a functional cell surface TWEAK-APRIL fusion protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

The Fn14 immediate-early response gene is induced during liver regeneration and highly expressed in both human and murine hepatocellular carcinomas

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

TNF-like weak inducer of apoptosis (TWEAK) induces inflammatory and proliferative effects in human kidney cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Proinflammatory effects of TWEAK/Fn14 interactions in glomerular mesangial cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

A novel TNF receptor family member binds TWEAK and is implicated in angiogenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Cytokine cooperation in renal tubular cell injury: the role of TWEAK.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Pathogenic autoantibodies in lupus nephritis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Considering TWEAK as a target for therapy in renal and vascular injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Urinary TWEAK as a biomarker of lupus nephritis: a multicenter cohort study.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

The two NF-kappaB activation pathways and their role in innate and adaptive immunity

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

I kappa B alpha promoter polymorphisms in patients with systemic lupus erythematosus.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Therapeutic potential of inhibition of the NF-kappaB pathway in the treatment of inflammation and cancer

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Cytokine gene transcription by NF-kappa B family members in patients with inflammatory bowel disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Bcl-2 down-regulates the activity of transcription factor NF-kappaB induced upon apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

NF-kappaB signaling pathways in mammalian and insect innate immunity

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

NF-kappaB regulation in the immune system

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

The TNF and TNF receptor superfamilies: integrating mammalian biology

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

NF-kappaB signalling in cerebral ischaemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Soluble and transmembrane TNF-like weak inducer of apoptosis differentially activate the classical and noncanonical NF-kappa B pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Tumor necrosis factor-related weak inducer of apoptosis augments matrix metalloproteinase 9 (MMP-9) production in skeletal muscle through the activation of nuclear factor-kappaB-inducing kinase and p38 mitogen-activated protein kinase: a potential r

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

TNF-like weak inducer of apoptosis (TWEAK) activates proinflammatory signaling pathways and gene expression through the activation of TGF-beta-activated kinase 1

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Pro-inflammatory effect of TWEAK/Fn14 interaction on human umbilical vein endothelial cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Pathogenesis of kidney disease in systemic lupus erythematosus

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Chemokines: therapeutic targets for autoimmune and inflammatory renal disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Gene transfer of RANTES elicits autoimmune renal injury in MRL-Fas(1pr) mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Antagonist of monocyte chemoattractant protein 1 ameliorates the initiation and progression of lupus nephritis and renal vasculitis in MRL/lpr mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Monocyte chemoattractant protein 1-dependent leukocytic infiltrates are responsible for autoimmune disease in MRL-Fas(lpr) mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Transient TWEAK overexpression leads to a general salivary epithelial cell proliferation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Apoptosis and proliferating cell nuclear antigen in lupus nephritis (class IV) and membranoproliferative glomerulonephritis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

TWEAK induces angiogenesis and proliferation of endothelial cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Conditional switching of vascular endothelial growth factor (VEGF) expression in tumors: induction of endothelial cell shedding and regression of hemangioblastoma-like vessels by VEGF withdrawal

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Multiple pathways of TWEAK-induced cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

TWEAK-Fn14 pathway inhibition protects the integrity of the neurovascular unit during cerebral ischemia.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Tumor necrosis factor-like weak inducer of apoptosis increases the permeability of the neurovascular unit through nuclear factor-kappa B pathway activation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

TWEAK-Fn14 interaction enhances plasminogen activator inhibitor 1 and tissue factor expression in atherosclerotic plaques and in cultured vascular smooth muscle cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00011-011-0420-8

Fibroblasts in mechanically stressed collagen lattices assume a "synthetic" phenotype

21 January 2018

1 reference

12 December 2020

[TWEAK, the facilitator of acute kidney injury]

1 reference

12 December 2020

Th1/Th2 balance of SLE patients with lupus nephritis.

1 reference

12 December 2020

Involvement of TNF-like weak inducer of apoptosis in the pathogenesis of collagen-induced arthritis

1 reference

12 December 2020

Alterations in TNF- and IL-related gene expression in space-flown WI38 human fibroblasts

1 reference

12 December 2020

TWEAK stimulation of kidney resident cells in the pathogenesis of graft versus host induced lupus nephritis

1 reference

12 December 2020

TWEAK/Fn14 interactions are instrumental in the pathogenesis of nephritis in the chronic graft-versus-host model of systemic lupus erythematosus

1 reference

12 December 2020

TWEAK activates the non-canonical NFkappaB pathway in murine renal tubular cells: modulation of CCL21

1 reference

12 December 2020

Down-regulated expression of TWEAK mRNA in acute and chronic inflammatory pathologies

1 reference

12 December 2020

In situ glomerular expression of activated NF-kappaB in human lupus nephritis and other non-proliferative proteinuric glomerulopathy

1 reference

12 December 2020

TWEAK stimulation of astrocytes and the proinflammatory consequences

1 reference

12 December 2020

TWEAK regulates proliferation and differentiation of adult neural progenitor cells

1 reference

12 December 2020

Increased expression of c-rel, from the NF-kappaB/Rel family, in T cells from patients with systemic lupus erythematosus

1 reference

12 December 2020

Gene expression of TWEAK/Fn14 and IP-10/CXCR3 in glomerulus and tubulointerstitium of patients with lupus nephritis

1 reference

12 December 2020

Multiple autoantibodies form the glomerular immune deposits in patients with systemic lupus erythematosus.

1 reference

12 December 2020

TWEAK is an endothelial cell growth and chemotactic factor that also potentiates FGF-2 and VEGF-A mitogenic activity

1 reference

12 December 2020

TWEAK is a novel arthritogenic mediator

1 reference

12 December 2020

Inhibition of the p50 (NKkappaB1) subunit of NF-kappaB by phosphorothioate-modified antisense oligodeoxynucleotides reduces NF-kappaB expression and immunoglobulin synthesis in murine B cells

1 reference

12 December 2020

10.1007/S00011-011-0420-8

Identifiers

DOI

1 reference

26 August 2017

1 reference

PubMed ID

22297307