(Q38001328)

26 August 2017

title

Essential roles of gangliosides in the formation and maintenance of membrane microdomains in brain tissues (English)

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26 August 2017

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Yuhsuke Ohmi

1

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26 August 2017

Yuki Ohkawa

2

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26 August 2017

Yoshio Yamauchi

3

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26 August 2017

Orie Tajima

4

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26 August 2017

Keiko Furukawa

5

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26 August 2017

Koichi Furukawa

6

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26 August 2017

publication date

10 April 2012

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26 August 2017

published in

Neurochemical Research

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26 August 2017

volume

37

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26 August 2017

issue

6

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26 August 2017

page(s)

1185-1191

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The role(s) of gangliosides in neural differentiation and repair: a perspective

26 August 2017

cites work

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

A vital role for glycosphingolipid synthesis during development and differentiation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Sensory nerve-dominant nerve degeneration and remodeling in the mutant mice lacking complex gangliosides.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Mice lacking complex gangliosides develop Wallerian degeneration and myelination defects

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Mice with disrupted GM2/GD2 synthase gene lack complex gangliosides but exhibit only subtle defects in their nervous system

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Reduced motor and sensory functions and emotional response in GM3-only mice: emergence from early stage of life and exacerbation with aging.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Cell-specific deletion of glucosylceramide synthase in brain leads to severe neural defects after birth.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Gangliosides play pivotal roles in the regulation of complement systems and in the maintenance of integrity in nerve tissues

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

The importance of inflammatory mechanisms in Alzheimer disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Neurons express proteins of the classical complement pathway in Alzheimer disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Complement activation by beta-amyloid in Alzheimer disease

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Functional rafts in cell membranes

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Complement in central nervous system inflammation

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Complement in Alzheimer's disease: opportunities for modulating protective and pathogenic events

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

The classical complement cascade mediates CNS synapse elimination

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Characterization of C3a and C5a receptors in rat cerebellar granule neurons during maturation. Neuroprotective effect of C5a against apoptotic cell death

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Complement: a novel factor in basal and ischemia-induced neurogenesis

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Prominent neurodegeneration and increased plaque formation in complement-inhibited Alzheimer's mice

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Up-regulated production and activation of the complement system in Alzheimer's disease brain

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Complement C5a receptor-mediated signaling may be involved in neurodegeneration in Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Myelin-associated glycoprotein and complementary axonal ligands, gangliosides, mediate axon stability in the CNS and PNS: neuropathology and behavioral deficits in single- and double-null mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Gangliosides potentially inhibit extracellular nucleotide metabolism

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Sphingolipids of the nucleus and their role in nuclear signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Effect of ganglioside and tetraspanins in microdomains on interaction of integrins with fibroblast growth factor receptor

21 January 2018

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Critical protection from renal ischemia reperfusion injury by CD55 and CD59.

21 January 2018

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Revitalizing membrane rafts: new tools and insights

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11064-012-0764-7

Refractory skin injury in complex knock-out mice expressing only the GM3 ganglioside

21 January 2018

1 reference

12 December 2020

Gangliosides are essential in the protection of inflammation and neurodegeneration via maintenance of lipid rafts: elucidation by a series of ganglioside-deficient mutant mice

1 reference

12 December 2020

b-series Ganglioside deficiency exhibits no definite changes in the neurogenesis and the sensitivity to Fas-mediated apoptosis but impairs regeneration of the lesioned hypoglossal nerve

1 reference

12 December 2020

Respective roles of decay-accelerating factor and CD59 in circumventing glomerular injury in acute nephrotoxic serum nephritis

1 reference

12 December 2020

Induction of decay-accelerating factor by cytokines or the membrane-attack complex protects vascular endothelial cells against complement deposition

1 reference

12 December 2020

GM1-ganglioside-mediated activation of the unfolded protein response causes neuronal death in a neurodegenerative gangliosidosis

1 reference

12 December 2020

Deficiency of complement defense protein CD59 may contribute to neurodegeneration in Alzheimer's disease

1 reference

12 December 2020

10.1007/S11064-012-0764-7

Identifiers

DOI

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Dimensions Publication ID

26 August 2017

1040569048

0 references

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