(Q38310551)

2 September 2017

title

Hypoxia induces p53-dependent transactivation and Fas/CD95-dependent apoptosis (English)

1 reference

2 September 2017

Liu T

1

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2 September 2017

Laurell C

2

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2 September 2017

Selivanova G

3

1 reference

2 September 2017

Lundeberg J

4

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2 September 2017

Nilsson P

5

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2 September 2017

Wiman KG

6

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2 September 2017

publication date

18 August 2006

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Cell Death & Differentiation

2 September 2017

published in

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2 September 2017

volume

14

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2 September 2017

issue

3

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2 September 2017

page(s)

411-421

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https://scigraph.springernature.com/pub.10.1038/sj.cdd.4402022

2 September 2017

exact match

0 references

cites work

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Hypoxia induces accumulation of p53 protein, but activation of a G1-phase checkpoint by low-oxygen conditions is independent of p53 status.

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Regulation of p53 by hypoxia: dissociation of transcriptional repression and apoptosis from p53-dependent transactivation.

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ATR/ATM targets are phosphorylated by ATR in response to hypoxia and ATM in response to reoxygenation

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p38 Mitogen-activated protein kinase mediates hypoxic regulation of Mdm2 and p53 in neurons

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Myc and E2F1 induce p53 through p14ARF-independent mechanisms in human fibroblasts

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p53 activates the CD95 (APO-1/Fas) gene in response to DNA damage by anticancer drugs

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inferred from DOI database lookup

7 January 2021

based on heuristic

cDNA sequence and chromosomal localization of a novel human protein, RBQ-1 (RBBP6), that binds to the retinoblastoma gene product

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4402022

inferred from DOI database lookup

7 January 2021

based on heuristic

Concepts in the use of TRAIL/Apo2L: an emerging biotherapy for myeloma and other neoplasias

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4402022

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/SJ.CDD.4402022

1 reference

Dimensions Publication ID

2 September 2017

1011232935

0 references

1 reference