(Q38357273)

English

Reciprocal effects of varicella-zoster virus (VZV) and AP1: activation of jun, fos and ATF-2 after VZV infection and their importance for the regulation of viral genes

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scholarly article

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Europe PubMed Central

PubMed publication ID

2 September 2017

Reciprocal effects of varicella-zoster virus (VZV) and AP1: activation of jun, fos and ATF-2 after VZV infection and their importance for the regulation of viral genes (English)

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

author name string

Markus Rahaus

1

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

Manfred H Wolff

2

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

publication date

1 March 2003

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

92

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

1

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

9-21

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

A rapid micropreparation technique for extraction of DNA-binding proteins from limiting numbers of mammalian cells

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Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factor

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Activation of Proto-Oncogenes: an Immediate Early Event in Human Cytomegalovirus Infection

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Cloning and characterization of a novel transcription factor involved in cellular proliferation arrest: PATF.

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The complete DNA sequence of varicella-zoster virus

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The 72K IE1 and 80K IE2 proteins of human cytomegalovirus independently trans-activate the c-fos, c-myc and hsp70 promoters via basal promoter elements

1 reference

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Cross-family dimerization of transcription factors Fos/Jun and ATF/CREB alters DNA binding specificity

1 reference

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Activation of the cellular transcription factor AP-1 in herpes simplex virus infected cells is dependent on the viral immediate-early protein ICPO

1 reference

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AP-1 function and regulation

1 reference

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Induction and repression of cellular gene transcription during herpes simplex virus infection are mediated by different viral immediate-early gene products

1 reference

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Accumulation of specific RNAs encoding transcriptional factors and stress response proteins against a background of severe depletion of cellular RNAs in cells infected with herpes simplex virus 1.

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Epstein-Barr virus-mediated B-cell proliferation is dependent upon latent membrane protein 1, which simulates an activated CD40 receptor

1 reference

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Human cytomegalovirus IE1 protein activates AP-1 through a cellular protein kinase(s)

1 reference

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Characterization of viremia at different stages of varicella-zoster virus infection

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The cellular transcription factor USF cooperates with varicella-zoster virus immediate-early protein 62 to symmetrically activate a bidirectional viral promoter

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Transcriptional regulation by cyclic AMP.

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Degradation of cellular mRNA during infection by herpes simplex virus

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Requirement of protein synthesis for the degradation of host mRNA in Friend erythroleukemia cells infected wtih herpes simplex virus type 1

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Selective inhibition of IL-2 gene expression by IL-2 antisense oligonucleotides blocks heart allograft rejection

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Influence of different cellular transcription factors on the regulation of varicella-zoster virus glycoproteins E (gE) and I (gI) UTR's activity

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Transcription factor Sp1 is involved in the regulation of varicella-zoster virus glycoprotein E.

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Herpes simplex virus-induced changes in cellular and adenovirus RNA metabolism in an adenovirus type 5-transformed human cell line

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Varicella-zoster virus (VZV) mediates a delayed host shutoff independent of open reading frame (ORF) 17 expression

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Hepatitis B virus HBx protein induces transcription factor AP-1 by activation of extracellular signal-regulated and c-Jun N-terminal mitogen-activated protein kinases

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Identifiers

10.1016/S0168-1702(02)00310-6

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

2 September 2017

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