(Q38386988)

2 September 2017

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title

Immunoglobulin A nephropathy: current progress and future directions (English)

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2 September 2017

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Chunlei Zhang

1

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2 September 2017

Xuehui Zeng

2

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2 September 2017

Zhongxin Li

3

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2 September 2017

Zhe Wang

4

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2 September 2017

Shunmin Li

5

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2 September 2017

4 March 2015

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2 September 2017

Translational Research

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2 September 2017

166

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2 September 2017

2

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2 September 2017

134-144

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Pathology of IgA nephropathy

2 September 2017

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

A tricontinental view of IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

The Oxford classification of IgA nephropathy: rationale, clinicopathological correlations, and classification

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

The Oxford classification of IgA nephropathy: pathology definitions, correlations, and reproducibility

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Henoch-Schonlein purpura nephritis: pathophysiology, treatment, and future strategy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Role of macromolecular IgA in IgA nephropathy

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Polymeric IgA1 from patients with IgA nephropathy upregulates transforming growth factor-beta synthesis and signal transduction in human mesangial cells via the renin-angiotensin system

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Differential glycosylation of polymeric and monomeric IgA: a possible role in glomerular inflammation in IgA nephropathy

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Dimeric and polymeric IgA, but not monomeric IgA, enhance the production of IL-6 by human renal mesangial cells

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

IgA nephropathy: molecular mechanisms of the disease

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Variants of C1GALT1 gene are associated with the genetic susceptibility to IgA nephropathy

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Interaction between variants of two glycosyltransferase genes in IgA nephropathy

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

B-cell O-galactosyltransferase activity, and expression of O-glycosylation genes in bone marrow in IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Site of catabolism of autologous and heterologous IgA in non-human primates

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Interactions of human mesangial cells with IgA and IgA-containing immune complexes

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Association of soluble CD89 levels with disease progression but not susceptibility in IgA nephropathy.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Integrin α1/β1 and α2/β1 as a receptor for IgA1 in human glomerular mesangial cells in IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Identification of the transferrin receptor as a novel immunoglobulin (Ig)A1 receptor and its enhanced expression on mesangial cells in IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Charge-dependent binding of polymeric IgA1 to human mesangial cells in IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Deposition of mannan binding protein and mannan binding protein-mediated complement activation in the glomeruli of patients with IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Baicalein reduces inflammatory process in a rodent model of diabetic retinopathy

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

SHARPIN is essential for cytokine production, NF-κB signaling, and induction of Th1 differentiation by dendritic cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Chronic proliferative dermatitis in Sharpin null mice: development of an autoinflammatory disease in the absence of B and T lymphocytes and IL4/IL13 signaling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Aberrantly glycosylated IgA molecules downregulate the synthesis and secretion of vascular endothelial growth factor in human mesangial cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Complement and the kidney: what the nephrologist needs to know in 2006?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Glomerular activation of the lectin pathway of complement in IgA nephropathy is associated with more severe renal disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

ACE-inhibitor suppresses the apoptosis induced by endoplasmic reticulum stress in renal tubular in experimental diabetic rats

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Autophagy inhibitors promoted aristolochic acid I induced renal tubular epithelial cell apoptosis via mitochondrial pathway but alleviated nonapoptotic cell death in mouse acute aritolochic acid nephropathy model

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

SHARPIN is a key regulator of immune and inflammatory responses

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Aberrant IgA1 glycosylation is inherited in familial and sporadic IgA nephropathy

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

HLA has strongest association with IgA nephropathy in genome-wide analysis

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Genome-wide association study identifies susceptibility loci for IgA nephropathy

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Geographic differences in genetic susceptibility to IgA nephropathy: GWAS replication study and geospatial risk analysis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

A genome-wide association study in Han Chinese identifies multiple susceptibility loci for IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Inherited IgA glycosylation pattern in IgA nephropathy and HSP nephritis: where do we go next?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Discovery of new risk loci for IgA nephropathy implicates genes involved in immunity against intestinal pathogens

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

The immune geography of IgA induction and function

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Global numbers of infection and disease burden of soil transmitted helminth infections in 2010.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Genome-wide association identifies multiple ulcerative colitis susceptibility loci

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

The histopathologic spectrum of kidney biopsies in patients with inflammatory bowel disease

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Oxidative stress and galactose-deficient IgA1 as markers of progression in IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Clinical and prognostic implications of serum uric acid levels on IgA nephropathy: a cohort study of 348 cases with a mean 5-year follow-up

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Activated complement C3: a potentially novel predictor of progressive IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Examination of the molecular diversity of alpha1 antitrypsin in urine: deficit of an alpha1 globulin fraction on cellulose acetate membrane electrophoresis.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

The ratio of epidermal growth factor to monocyte chemotactic peptide-1 in the urine predicts renal prognosis in IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Urinary mannose-binding lectin is a biomarker for predicting the progression of immunoglobulin (Ig)A nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Urine protein patterns can serve as diagnostic tools in patients with IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Electrophoretic methods for analysis of urinary polypeptides in IgA-associated renal diseases

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Urinary biomarkers of IgA nephropathy and other IgA-associated renal diseases

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

MicroRNAs in IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Genome-wide analysis of microRNAs expression profiling in patients with primary IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Abnormal miR-148b expression promotes aberrant glycosylation of IgA1 in IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

miR-29c is downregulated in renal interstitial fibrosis in humans and rats and restored by HIF-α activation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Intrarenal expression of microRNAs in patients with IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Elevated levels of miR-146a and miR-155 in kidney biopsy and urine from patients with IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Urinary miR-21, miR-29, and miR-93: novel biomarkers of fibrosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

A panel of serum biomarkers differentiates IgA nephropathy from other renal diseases

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

MicroRNAs: small RNAs with a big role in gene regulation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Urinary interleukin-6 (IL-6) and transforming growth factor (TGF-β) levels in corticosteroidtreated patients with IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Expression and significance of TGF-beta1/Smad signaling pathway in children with IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Micro-RNA expression in the urinary sediment of patients with chronic kidney diseases

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Expression of microRNAs in the urinary sediment of patients with IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Fibroblast growth factor-23 and calcium phosphate product in young chronic kidney disease patients: a cross-sectional study

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Soluble transferrin receptor in urine, a new biomarker for IgA nephropathy and Henoch-Schönlein purpura nephritis.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Current therapy for IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Antithymocyte globulin (ATG) induction therapy and disease recurrence in renal transplant recipients with primary IgA nephropathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

Steroids and recurrent IgA nephropathy after kidney transplantation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRSL.2015.02.007

10.1016/J.TRSL.2015.02.007

7 January 2021

Identifiers

DOI

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2 September 2017

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