(Q38459491)

3 September 2017

title

Oxidative stress and HNE conjugation of GLUT3 are increased in the hippocampus of diabetic rats subjected to stress (English)

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3 September 2017

L P Reagan

1

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3 September 2017

A M Magariños

2

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3 September 2017

D K Yee

3

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3 September 2017

L I Swzeda

4

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3 September 2017

A Van Bueren

5

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3 September 2017

A L McCall

6

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3 September 2017

B S McEwen

7

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3 September 2017

language of work or name

English

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publication date

1 April 2000

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3 September 2017

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3 September 2017

volume

862

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3 September 2017

issue

1-2

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3 September 2017

page(s)

292-300

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Dehydroepiandrosterone administration prevents the oxidative damage induced by acute hyperglycemia in rats.

3 September 2017

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Metabolic control may influence the increased superoxide generation in diabetic serum.

7 January 2021

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7 January 2021

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Local Cerebral Glucose Utilization Is Increased in Acutely Adrenalectomized Rats

7 January 2021

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4-Hydroxynonenal, an aldehydic product of membrane lipid peroxidation, impairs glutamate transport and mitochondrial function in synaptosomes.

7 January 2021

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Effect of diabetes on levels of lipid peroxides and glycolipids in rat brain

7 January 2021

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7 January 2021

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Modification of ion homeostasis by lipid peroxidation: roles in neuronal degeneration and adaptive plasticity

7 January 2021

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Metabolic fuel and amino acid transport into the brain in experimental diabetes mellitus

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Immunohistochemical localization of the neuron-specific glucose transporter (GLUT3) to neuropil in adult rat brain

7 January 2021

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Immunological evidence for increased oxidative stress in diabetic rats

7 January 2021

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Modification of histidine residues in proteins by reaction with 4-hydroxynonenal

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Repeated restraint stress facilitates fear conditioning independently of causing hippocampal CA3 dendritic atrophy

7 January 2021

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inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/S0006-8993(00)02212-5

1 reference

3 September 2017

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