(Q38519525)

English

In vitro cellular aging is associated with enhanced proliferative capacity, G1 cell cycle modulation, and matrix metalloproteinase-9 regulation in mouse aortic smooth muscle cells

scientific article published on October 2003

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scholarly article

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Europe PubMed Central

PubMed publication ID

3 September 2017

In vitro cellular aging is associated with enhanced proliferative capacity, G1 cell cycle modulation, and matrix metalloproteinase-9 regulation in mouse aortic smooth muscle cells (English)

1 reference

Europe PubMed Central

PubMed publication ID

3 September 2017

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author name string

Sung-Kwon Moon

1

1 reference

Europe PubMed Central

PubMed publication ID

3 September 2017

Byung-Yoon Cha

2

1 reference

Europe PubMed Central

PubMed publication ID

3 September 2017

Cheorl-Ho Kim

3

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Europe PubMed Central

PubMed publication ID

3 September 2017

language of work or name

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publication date

1 October 2003

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PubMed publication ID

3 September 2017

number of pages

10

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Archives of Biochemistry and Biophysics

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Europe PubMed Central

PubMed publication ID

3 September 2017

418

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Europe PubMed Central

PubMed publication ID

3 September 2017

1

1 reference

Europe PubMed Central

PubMed publication ID

3 September 2017

39-48

1 reference

Europe PubMed Central

PubMed publication ID

3 September 2017

Atherosclerosis — An Inflammatory Disease

1 reference

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Physiology of cardiovascular aging

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Cardiovascular regulatory mechanisms in advanced age.

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Enhanced proliferation and migration and altered cytoskeletal proteins in early passage smooth muscle cells from young and old rat aortic explants

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Aging and arteriosclerosis. The increased proliferation of arterial smooth muscle cells isolated from old rats is associated with increased platelet-derived growth factor-like activity

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7 January 2021

Vascular smooth muscle cell outgrowth, proliferation, and apoptosis in young and old rats

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7 January 2021

Age-related loss of proliferative activity of human vascular smooth muscle cells in culture

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Cell proliferation in human coronary arteries

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The pathogenesis of atherosclerosis: a perspective for the 1990s

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G1 phase progression: cycling on cue

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Cancer cell cycles

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Matrix metalloproteinase-9 is necessary for the regulation of smooth muscle cell replication and migration after arterial injury

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Targeted disruption of the matrix metalloproteinase-9 gene impairs smooth muscle cell migration and geometrical arterial remodeling.

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Cytokine-stimulated human vascular smooth muscle cells synthesize a complement of enzymes required for extracellular matrix digestion

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Quercetin exerts multiple inhibitory effects on vascular smooth muscle cells: role of ERK1/2, cell-cycle regulation, and matrix metalloproteinase-9.

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7 January 2021

Mitogen-activated protein kinases mediate matrix metalloproteinase-9 expression in vascular smooth muscle cells

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Isolation of gene markers of differentiated and proliferating vascular smooth muscle cells.

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Molecular control of vascular smooth muscle cell differentiation

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Molecular mechanisms in intimal hyperplasia

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Increased expression of matrix metalloproteinase-2 in the thickened intima of aged rats

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Replicative senescence: implications for in vivo aging and tumor suppression

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Histopathology of in-stent restenosis in patients with peripheral artery disease

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Age-dependent increase in c-fos activity and cyclin A expression in vascular smooth muscle cells. A potential link between aging, smooth muscle cell proliferation and atherosclerosis

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The emerging concept of vascular remodeling

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Divergent regulation by growth factors and cytokines of 95 kDa and 72 kDa gelatinases and tissue inhibitors or metalloproteinases-1, -2, and -3 in rabbit aortic smooth muscle cells

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7 January 2021

Matrix metalloproteinases and cardiovascular disease

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7 January 2021

Matrix metalloproteinase-9 overexpression enhances vascular smooth muscle cell migration and alters remodeling in the injured rat carotid artery

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7 January 2021

Localization of stromelysin gene expression in atherosclerotic plaques by in situ hybridization

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7 January 2021

Migration of cultured vascular smooth muscle cells through a basement membrane barrier requires type IV collagenase activity and is inhibited by cellular differentiation

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7 January 2021

Synergistic upregulation of metalloproteinase-9 by growth factors and inflammatory cytokines: an absolute requirement for transcription factor NF-kappa B

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7 January 2021

Heterogeneous smooth muscle cell population derived from small and larger arteries.

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7 January 2021

Propidium iodide as a nuclear counterstain for immunofluorescence studies on cells in culture

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7 January 2021

p21 is a universal inhibitor of cyclin kinases

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p27, a novel inhibitor of G1 cyclin-Cdk protein kinase activity, is related to p21

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p21(Waf1/Cip1) is an assembly factor required for platelet-derived growth factor-induced vascular smooth muscle cell proliferation

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New functional activities for the p21 family of CDK inhibitors

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p21-containing cyclin kinases exist in both active and inactive states

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Expressional regulation of smooth muscle cell-specific genes in association with phenotypic modulation

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7 January 2021

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Identifiers

10.1016/S0003-9861(03)00402-8

1 reference

Europe PubMed Central

PubMed publication ID

3 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

3 September 2017

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