(Q38941672)

7 September 2017

title

Angiomotin decreases lung cancer progression by sequestering oncogenic YAP/TAZ and decreasing Cyr61 expression (English)

1 reference

7 September 2017

1 reference

Y-L Hsu

1

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7 September 2017

J-Y Hung

2

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7 September 2017

S-H Chou

3

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7 September 2017

M-S Huang

4

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7 September 2017

M-J Tsai

5

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7 September 2017

Y-S Lin

6

1 reference

7 September 2017

S-Y Chiang

7

1 reference

7 September 2017

Y-W Ho

8

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7 September 2017

C-Y Wu

9

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7 September 2017

P-L Kuo

10

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7 September 2017

language of work or name

English

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publication date

10 November 2014

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7 September 2017

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7 September 2017

volume

34

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7 September 2017

issue

31

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7 September 2017

page(s)

4056-4068

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https://scigraph.springernature.com/pub.10.1038/onc.2014.333

7 September 2017

exact match

0 references

cites work

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Angiomotin regulates endothelial cell-cell junctions and cell motility

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Phosphorylation of angiomotin by Lats1/2 kinases inhibits F-actin binding, cell migration, and angiogenesis

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A tight junction-associated Merlin-angiomotin complex mediates Merlin's regulation of mitogenic signaling and tumor suppressive functions

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Serum deprivation inhibits the transcriptional co-activator YAP and cell growth via phosphorylation of the 130-kDa isoform of Angiomotin by the LATS1/2 protein kinases

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YAP promotes breast cell proliferation and survival partially through stabilizing the KLF5 transcription factor

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Yes-associated protein (YAP) transcriptional coactivator functions in balancing growth and differentiation in skin

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A DNA vaccine targeting angiomotin inhibits angiogenesis and suppresses tumor growth

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Angiomotin is a novel Hippo pathway component that inhibits YAP oncoprotein

21 January 2018

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Matricellular protein Cyr61 bridges lysophosphatidic acid and integrin pathways leading to cell migration.

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PubMed

https://pubmed.ncbi.nlm.nih.gov/25381822

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Expression of Yes-associated protein in non-small cell lung cancer and its relationship with clinical pathological factors

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/25381822

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1038/ONC.2014.333

1 reference

Dimensions Publication ID

7 September 2017

1003297849

0 references

1 reference