(Q39003629)

English

KLF9, a transcription factor induced in flutamide-caused cell apoptosis, inhibits AKT activation and suppresses tumor growth of prostate cancer cells

scientific article published on 16 April 2014

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scholarly article

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Europe PubMed Central

PubMed publication ID

8 September 2017

KLF9, a transcription factor induced in flutamide-caused cell apoptosis, inhibits AKT activation and suppresses tumor growth of prostate cancer cells (English)

1 reference

Europe PubMed Central

PubMed publication ID

8 September 2017

prostate cancer

1 reference

based on heuristic

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author name string

Pengliang Shen

1

1 reference

Europe PubMed Central

PubMed publication ID

8 September 2017

Jiabin Sun

2

1 reference

Europe PubMed Central

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8 September 2017

Guiqin Xu

3

1 reference

Europe PubMed Central

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8 September 2017

Li Zhang

4

1 reference

Europe PubMed Central

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8 September 2017

Zhaojuan Yang

5

1 reference

Europe PubMed Central

PubMed publication ID

8 September 2017

Suhua Xia

6

1 reference

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8 September 2017

Yang Wang

7

1 reference

Europe PubMed Central

PubMed publication ID

8 September 2017

Yongzhong Liu

8

1 reference

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8 September 2017

Guowei Shi

9

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8 September 2017

publication date

16 April 2014

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8 September 2017

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Europe PubMed Central

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8 September 2017

74

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8 September 2017

9

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8 September 2017

946-958

1 reference

Europe PubMed Central

PubMed publication ID

8 September 2017

Global cancer statistics

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Endocrine prevention and treatment of prostate cancer

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Effects of the antiandrogen flutamide on the expression of protein kinase C isoenzymes in LNCaP and PC3 human prostate cancer cells.

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

The role of antiandrogen monotherapy in the treatment of prostate cancer

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Survivin mediates resistance to antiandrogen therapy in prostate cancer

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

The development of androgen-independent prostate cancer

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Mammalian Krüppel-like factors in health and diseases

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Chromosomal localization and cDNA sequence of human BTEB, a GC box binding protein

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Krüppel-like factor 9 is a circadian transcription factor in human epidermis that controls proliferation of keratinocytes

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Krüppel-like factor KLF9 regulates PPARγ transactivation at the middle stage of adipogenesis

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Downregulation of Krüppel-like factor 9 in human colorectal cancer.

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Krüppel-like family of transcription factor 9, a differentiation-associated transcription factor, suppresses Notch1 signaling and inhibits glioblastoma-initiating stem cells

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Prognostic value and function of KLF4 in prostate cancer: RNAa and vector-mediated overexpression identify KLF4 as an inhibitor of tumor cell growth and migration

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

KLF5 is frequently deleted and down-regulated but rarely mutated in prostate cancer

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

KLF6 induces apoptosis in prostate cancer cells through up-regulation of ATF3

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Krüppel-like factor 8 is a novel androgen receptor co-activator in human prostate cancer

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

KLF6, a candidate tumor suppressor gene mutated in prostate cancer

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Characterization of the prostate cancer susceptibility gene KLF6 in human and mouse prostate cancers.

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

The emerging role of Krüppel-like factors in endocrine-responsive cancers of female reproductive tissues

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Akt signaling and cancer: surviving but not moving on.

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Targeting the PI3K/AKT pathway for the treatment of prostate cancer

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Androgen receptor antagonist versus agonist activities of the fungicide vinclozolin relative to hydroxyflutamide

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

KLF9 is a novel transcriptional regulator of bortezomib- and LBH589-induced apoptosis in multiple myeloma cells

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Kruppel-like factor 9 is a negative regulator of ligand-dependent estrogen receptor alpha signaling in Ishikawa endometrial adenocarcinoma cells

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Bortezomib represses HIF-1α protein expression and nuclear accumulation by inhibiting both PI3K/Akt/TOR and MAPK pathways in prostate cancer cells

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Bortezomib-mediated inhibition of steroid receptor coactivator-3 degradation leads to activated Akt.

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Vorinostat and bortezomib synergistically cause ubiquitinated protein accumulation in prostate cancer cells

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Bortezomib reverses the proliferative and antiapoptotic effect of neuropeptides on prostate cancer cells

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Global expression changes of constitutive and hormonally regulated genes during endometrial neoplastic transformation

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Antagonists of growth hormone-releasing hormone inhibit growth of androgen-independent prostate cancer through inactivation of ERK and Akt kinases

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

HER-2/neu blocks tumor necrosis factor-induced apoptosis via the Akt/NF-kappaB pathway

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Increased AKT activity contributes to prostate cancer progression by dramatically accelerating prostate tumor growth and diminishing p27Kip1 expression

1 reference

https://api.crossref.org/works/10.1002%2FPROS.22812

21 January 2018

Identifiers

10.1002/PROS.22812

1 reference

Europe PubMed Central

PubMed publication ID

8 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

8 September 2017

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