(Q39114231)

9 September 2017

title

Glucose-regulated protein 78 mediates hormone-independent prostate cancer progression and metastasis through maspin and COX-2 expression (English)

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9 September 2017

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Chun-Te Wu

1

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9 September 2017

Wen-Ching Wang

2

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9 September 2017

Miao-Fen Chen

3

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9 September 2017

Hou-Yu Su

4

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9 September 2017

Wei-Yu Chen

5

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9 September 2017

Chih-Hsiung Wu

6

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9 September 2017

Yu-Jia Chang

7

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9 September 2017

Hui-Hsiung Liu

8

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9 September 2017

publication date

7 August 2013

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9 September 2017

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9 September 2017

volume

35

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9 September 2017

issue

1

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9 September 2017

page(s)

195-204

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https://scigraph.springernature.com/pub.10.1007/s13277-013-1024-4

9 September 2017

exact match

0 references

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Coupling endoplasmic reticulum stress to the cell death program: role of the ER chaperone GRP78

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Stress protein GRP78 prevents apoptosis induced by calcium ionophore, ionomycin, but not by glycosylation inhibitor, tunicamycin, in human prostate cancer cells

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Vascular targeting and antiangiogenesis agents induce drug resistance effector GRP78 within the tumor microenvironment

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Activation of the ATF6, XBP1 and grp78 genes in human hepatocellular carcinoma: a possible involvement of the ER stress pathway in hepatocarcinogenesis

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De-regulation of GRP stress protein expression in human breast cancer cell lines.

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Fingerprinting the circulating repertoire of antibodies from cancer patients

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Glucose-regulated protein GRP78 is up-regulated in prostate cancer and correlates with recurrence and survival.

21 January 2018

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GRP78 up-regulation is associated with androgen receptor status, Hsp70-Hsp90 client proteins and castrate-resistant prostate cancer

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Maspin (SERPINB5) is an obligate intracellular serpin

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Role of maspin in cancer

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Maspin functions as tumor suppressor by increasing cell adhesion to extracellular matrix in prostate tumor cells

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G-helix of maspin mediates effects on cell migration and adhesion

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Maspin reduces prostate cancer metastasis to bone

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Maspin expression inhibits osteolysis, tumor growth, and angiogenesis in a model of prostate cancer bone metastasis

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Maspin augments proteasome inhibitor-induced apoptosis in prostate cancer cells

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Membrane nanotubes physically connect T cells over long distances presenting a novel route for HIV-1 transmission

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Glucose-regulated protein 78 (GRP78) silencing enhances cell migration but does not influence cell proliferation in hepatocellular carcinoma

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21 January 2018

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Blockade of store-operated Ca(2+) entry inhibits hepatocarcinoma cell migration and invasion by regulating focal adhesion turnover

21 January 2018

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Bisphenol A stimulates human prostate cancer cell migration via remodelling of calcium signalling.

21 January 2018

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Involvement of store-operated calcium signaling in EGF-mediated COX-2 gene activation in cancer cells.

21 January 2018

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21 January 2018

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The ER function BiP is a master regulator of ER function

21 January 2018

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12 December 2020

COX-2 promotes breast cancer cell radioresistance via p38/MAPK-mediated cellular anti-apoptosis and invasiveness

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12 December 2020

Cancer statistics, 2005

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12 December 2020

Drug resistance mediated by cellular stress response to the microenvironment of solid tumors

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12 December 2020

Glucose regulated protein 78: a critical link between tumor microenvironment and cancer hallmarks

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12 December 2020

Vitamin E inhibits the high-fat diet promoted growth of established human prostate LNCaP tumors in nude mice

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12 December 2020

Comparative proteomic analysis of the function and network mechanisms of MASPIN in human lung cells

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12 December 2020

Maspin is an angiogenesis inhibitor

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12 December 2020

Orai1/CRACM1 overexpression suppresses cell proliferation via attenuation of the store-operated calcium influx-mediated signalling pathway in A549 lung cancer cells

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12 December 2020

Survivin-mediated therapeutic efficacy of gemcitabine through glucose-regulated protein 78 in hepatocellular carcinoma

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12 December 2020

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12 December 2020

Cancer screening in men

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12 December 2020

10.1007/S13277-013-1024-4

Identifiers

DOI

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9 September 2017

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