(Q39356827)

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IGFBP-3 methylation-derived deficiency mediates the resistance to cisplatin through the activation of the IGFIR/Akt pathway in non-small cell lung cancer.

scientific article published on 30 April 2012

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30 January 2020

IGFBP-3 methylation-derived deficiency mediates the resistance to cisplatin through the activation of the IGFIR/Akt pathway in non-small cell lung cancer (English)

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30 January 2020

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Cristobal Belda Iniesta

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30 January 2020

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M Cortés-Sempere

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30 January 2020

M P de Miguel

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30 January 2020

O Pernía

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30 January 2020

C Rodriguez

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30 January 2020

J de Castro Carpeño

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30 January 2020

M Nistal

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30 January 2020

E Conde

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30 January 2020

F López-Ríos

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30 January 2020

R Perona

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30 January 2020

I Ibanez de Caceres

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30 April 2012

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30 January 2020

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30 January 2020

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30 January 2020

1274-1283

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30 January 2020

https://scigraph.springernature.com/pub.10.1038/onc.2012.146

0 references

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1 reference

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Implication of the insulin-like growth factor-IR pathway in the resistance of non-small cell lung cancer cells to treatment with gefitinib

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Heterodimerization of insulin-like growth factor receptor/epidermal growth factor receptor and induction of survivin expression counteract the antitumor action of erlotinib.

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21 January 2018

IGFBP-3 hypermethylation-derived deficiency mediates cisplatin resistance in non-small-cell lung cancer.

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Type 1 insulin-like growth factor receptor translocates to the nucleus of human tumor cells

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Drug-induced DNA hypermethylation: a potential mediator of acquired drug resistance during cancer chemotherapy

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Oxidative stress modulates DNA methylation during melanocyte anchorage blockade associated with malignant transformation.

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MicroRNA-29 family reverts aberrant methylation in lung cancer by targeting DNA methyltransferases 3A and 3B

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Inhibition of DNA methyltransferase reverses cisplatin induced drug resistance in murine neuroblastoma cells.

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Cyclical regulation of the insulin-like growth factor binding protein 3 gene in response to 1alpha,25-dihydroxyvitamin D3.

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Targeting histone deacetylases for the treatment of disease

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Hypoxic stress induces dimethylated histone H3 lysine 9 through histone methyltransferase G9a in mammalian cells

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The von Hippel-Lindau tumor suppressor protein regulates gene expression and tumor growth through histone demethylase JARID1C.

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Epigenetic regulation of X-linked cancer/germline antigen genes by DNMT1 and DNMT3b

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Patterns of somatic mutation in human cancer genomes

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PTEN/MMAC1 mutations identified in small cell, but not in non-small cell lung cancers

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MKP1/CL100 controls tumor growth and sensitivity to cisplatin in non-small-cell lung cancer.

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Overexpression of miR-200c induces chemoresistance in esophageal cancers mediated through activation of the Akt signaling pathway

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Insulin-like growth factor type-I receptor internalization and recycling mediate the sustained phosphorylation of Akt.

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The Grb10/Nedd4 complex regulates ligand-induced ubiquitination and stability of the insulin-like growth factor I receptor

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Randomized phase III trial of platinum-doublet chemotherapy followed by gefitinib compared with continued platinum-doublet chemotherapy in Japanese patients with advanced non-small-cell lung cancer: results of a west Japan thoracic oncology group tr

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Antagonism between gefitinib and cisplatin in non-small cell lung cancer cells: why randomized trials failed?

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High predictive value of epidermal growth factor receptor phosphorylation but not of EGFRvIII mutation in resected stage I non-small cell lung cancer (NSCLC)

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Candidate DNA methylation drivers of acquired cisplatin resistance in ovarian cancer identified by methylome and expression profiling

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