(Q39418101)

11 September 2017

title

TGFβ modulates cell-to-cell communication in early epithelial-to-mesenchymal transition (English)

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11 September 2017

4

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11 September 2017

C E Hills

1

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11 September 2017

E Siamantouras

2

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11 September 2017

S W Smith

3

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11 September 2017

K-K Liu

5

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11 September 2017

P E Squires

6

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11 September 2017

publication date

4 January 2012

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11 September 2017

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11 September 2017

volume

55

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11 September 2017

issue

3

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11 September 2017

page(s)

812-824

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https://scigraph.springernature.com/pub.10.1007/s00125-011-2409-9

11 September 2017

exact match

0 references

cites work

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TGF-beta1-induced epithelial-to-mesenchymal transition and therapeutic intervention in diabetic nephropathy.

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Diabetic nephropathy: mechanisms of renal disease progression

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Advanced glycation end products cause epithelial-myofibroblast transdifferentiation via the receptor for advanced glycation end products (RAGE)

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High glucose induces macrophage inflammatory protein-3 alpha in renal proximal tubule cells via a transforming growth factor-beta 1 dependent mechanism

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Structural-functional relationships in diabetic nephropathy

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Phosphatidylinositol 3-kinase function is required for transforming growth factor beta-mediated epithelial to mesenchymal transition and cell migration.

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Integrity of cell-cell contacts is a critical regulator of TGF-beta 1-induced epithelial-to-myofibroblast transition: role for beta-catenin

21 January 2018

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Disruption of E-cadherin by matrix metalloproteinase directly mediates epithelial-mesenchymal transition downstream of transforming growth factor-beta1 in renal tubular epithelial cells.

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Biophysical evidence that connexin-36 forms functional gap junction channels between pancreatic mouse beta-cells

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C-cadherin ectodomain structure and implications for cell adhesion mechanisms

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Differential phosphorylation of the gap junction protein connexin43 in junctional communication-competent and -deficient cell lines

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Construction of epithelioid sheets by transfection of mouse sarcoma cells with cDNAs for chicken cell adhesion molecules

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Reduced connexin 43 expression and its effect on the development of vascular lesions in retinas of diabetic mice

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High glucose-induced downregulation of connexin 43 expression promotes apoptosis in microvascular endothelial cells.

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Differential connexin expression in preglomerular and postglomerular vasculature: accentuation during diabetes

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Increased eNOS accounts for changes in connexin expression in renal arterioles during diabetes

21 January 2018

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High glucose alters connexin 43 expression and gap junction intercellular communication activity in retinal pericytes

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Downregulation of connexin 43 expression by high glucose reduces gap junction activity in microvascular endothelial cells.

21 January 2018

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High glucose down-regulates intercellular communication in retinal endothelial cells by enhancing degradation of connexin 43 by a proteasome-dependent mechanism

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High glucose up-regulates ENaC and SGK1 expression in HCD-cells.

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Glucose-evoked alterations in connexin43-mediated cell-to-cell communication in human collecting duct: a possible role in diabetic nephropathy.

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TGF-beta1-mediated alterations of renal proximal tubular epithelial cell phenotype

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Proinsulin C-peptide antagonizes the profibrotic effects of TGF-beta1 via up-regulation of retinoic acid and HGF-related signaling pathways

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21 January 2018

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Transforming growth factor-{beta}1 induces Smad3-dependent {beta}1 integrin gene expression in epithelial-to-mesenchymal transition during chronic tubulointerstitial fibrosis

21 January 2018

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A tale of two proteins: differential roles and regulation of Smad2 and Smad3 in TGF-beta signaling

21 January 2018

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Downregulation of SnoN expression in obstructive nephropathy is mediated by an enhanced ubiquitin-dependent degradation.

21 January 2018

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Smad7 inhibits fibrotic effect of TGF-Beta on renal tubular epithelial cells by blocking Smad2 activation

21 January 2018

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The role of TGF-β and epithelial-to mesenchymal transition in diabetic nephropathy.

21 January 2018

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12 December 2020

High glucose-induced hypertrophy of mesangial cells is reversed by connexin43 overexpression via PTEN/Akt/mTOR signaling.

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12 December 2020

Dynamics of connexins, E-cadherin and alpha-catenin on cell membranes during gap junction formation

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12 December 2020

Monoclonal antibody ECCD-1 inhibits intercellular communication in teratocarcinoma PCC3 cells

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12 December 2020

Molecular insights into renal interstitial fibrosis

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12 December 2020

Mesangial expansion as a central mechanism for loss of kidney function in diabetic patients

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12 December 2020

Essential role for Smad3 in angiotensin II-induced tubular epithelial-mesenchymal transition.

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12 December 2020

Hyperglycemia and diabetic kidney disease. The case for transforming growth factor-beta as a key mediator

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12 December 2020

Early role of Fsp1 in epithelial-mesenchymal transformation

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12 December 2020

Resolved: EMT produces fibroblasts in the kidney

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12 December 2020

Beta-cell crosstalk: a further dimension in the stimulus-secretion coupling of glucose-induced insulin release

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12 December 2020

C-peptide reverses TGF-beta1-induced changes in renal proximal tubular cells: implications for treatment of diabetic nephropathy

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12 December 2020

Altered gap junctional communication and renal haemodynamics in Zucker fatty rat model of type 2 diabetes

1 reference

12 December 2020

10.1007/S00125-011-2409-9

Identifiers

DOI

1 reference

11 September 2017

1 reference