(Q39602506)

English

Per-1 is a specific clock gene regulated by parathyroid hormone (PTH) signaling in osteoblasts and is functional for the transcriptional events induced by PTH.

scientific article published on February 2011

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

Per-1 is a specific clock gene regulated by parathyroid hormone (PTH) signaling in osteoblasts and is functional for the transcriptional events induced by PTH. (English)

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

0 references

author name string

Ryo Hanyu

1

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

Tadayoshi Hayata

2

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

Masashi Nagao

3

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

Yoshitomo Saita

4

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

Hiroaki Hemmi

5

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

Takuya Notomi

6

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

Tetsuya Nakamoto

7

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

Ernestina Schipani

8

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

Henry Knonenbery

9

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

Kazuo Kaneko

10

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

Hisashi Kurosawa

11

1 reference

Europe PubMed Central

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13 September 2017

Yoichi Ezura

12

1 reference

Europe PubMed Central

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13 September 2017

Masaki Noda

13

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13 September 2017

publication date

1 February 2011

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Europe PubMed Central

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13 September 2017

Journal of Cellular Biochemistry

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13 September 2017

112

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13 September 2017

2

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13 September 2017

433-438

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

The use of PTH in the treatment of osteoporosis

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Orthopedic uses of teriparatide

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Activated parathyroid hormone/parathyroid hormone-related protein receptor in osteoblastic cells differentially affects cortical and trabecular bone

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Osteoblasts clock in for their day job.

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Back to the future: revisiting parathyroid hormone and calcitonin control of bone remodeling

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

The circadian modulation of leptin-controlled bone formation.

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Molecular biology of bone remodeling: implications for new therapeutic targets for osteoporosis.

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Post-translational modifications regulate the ticking of the circadian clock.

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Circadian rhythms, adrenergic hormones and trafficking of hematopoietic stem cells.

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Biochemical markers of bone turnover.

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Up-regulation of per mRNA expression by parathyroid hormone through a protein kinase A-CREB-dependent mechanism in chondrocytes.

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

PPARγ: a circadian transcription factor in adipogenesis and osteogenesis

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Unloading induces osteoblastic cell suppression and osteoclastic cell activation to lead to bone loss via sympathetic nervous system

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

The nucleocytoplasmic shuttling protein CIZ reduces adult bone mass by inhibiting bone morphogenetic protein-induced bone formation

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Osteopontin negatively regulates parathyroid hormone receptor signaling in osteoblasts

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Constitutively active parathyroid hormone receptor signaling in cells in osteoblastic lineage suppresses mechanical unloading-induced bone resorption

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Structural organization and biological relevance of oscillatory parathyroid hormone secretion

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Harnessing the parathyroids to create stronger bones

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Investigational anabolic therapies for osteoporosis

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

G Protein and its signaling pathway in bone development and disease

1 reference

https://api.crossref.org/works/10.1002%2FJCB.22957

21 January 2018

Identifiers

10.1002/JCB.22957

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

13 September 2017

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