(Q39775968)

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CFTR mediates apoptotic volume decrease and cell death by controlling glutathione efflux and ROS production in cultured mice proximal tubules.

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12 February 2020

CFTR mediates apoptotic volume decrease and cell death by controlling glutathione efflux and ROS production in cultured mice proximal tubules (English)

1 reference

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19906953%20AND%20SRC:MED&resulttype=core&format=json

12 February 2020

apoptotic process

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Isabelle Rubera

6

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12 February 2020

Baharia Mograbi

8

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12 February 2020

9

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12 February 2020

author name string

Sebastien l'Hoste

1

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12 February 2020

Abderrahmen Chargui

2

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12 February 2020

Radia Belfodil

3

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12 February 2020

Elisabeth Corcelle

4

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12 February 2020

Christophe Duranton

5

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12 February 2020

Chantal Poujeol

7

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12 February 2020

Philippe Poujeol

10

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12 February 2020

publication date

11 November 2009

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12 February 2020

American Journal of Physiology - Renal Physiology

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12 February 2020

298

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12 February 2020

F435-53

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12 February 2020

2

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12 February 2020

New benzo(b)thiophenesulphonamide 1,1-dioxide derivatives induce a reactive oxygen species-mediated process of apoptosis in tumour cells.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Role of TASK2 potassium channels regarding volume regulation in primary cultures of mouse proximal tubules.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

CFTR null mutation altered cAMP-sensitive and swelling-activated Cl- currents in primary cultures of mouse nephron.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

CFTR modulates programmed cell death by decreasing intracellular pH in Chinese hamster lung fibroblasts

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Swelling-activated chloride and potassium conductance in primary cultures of mouse proximal tubules. Implication of KCNE1 protein.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

CFTR-dependent and -independent swelling-activated K+ currents in primary cultures of mouse nephron.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Cell shrinkage and monovalent cation fluxes: role in apoptosis

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

cAMP signaling cascades and CFTR: is there more to learn?

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Role of actin filament organization in CFTR activation.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

The role of GSH efflux in staurosporine-induced apoptosis in colonic epithelial cells

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Oxidant stress stimulates anion secretion from the human airway epithelial cell line Calu-3: implications for cystic fibrosis lung disease

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Mutant cystic fibrosis transmembrane conductance regulator inhibits acidification and apoptosis in C127 cells: possible relevance to cystic fibrosis

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Determination of glutathione and glutathione disulfide using glutathione reductase and 2-vinylpyridine

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Glutathione export during apoptosis requires functional multidrug resistance-associated proteins

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Glutathione levels and BAX activation during apoptosis due to oxidative stress in cells expressing wild-type and mutant cystic fibrosis transmembrane conductance regulator

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

A role for CFTR in the elevation of glutathione levels in the lung by oral glutathione administration

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

CFTR directly mediates nucleotide-regulated glutathione flux.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Extracellular pH alkalinization by Cl-/HCO3- exchanger is crucial for TASK2 activation by hypotonic shock in proximal cell lines from mouse kidney.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

CFTR mediates cadmium-induced apoptosis through modulation of ROS level in mouse proximal tubule cells.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Role of TASK2 in the control of apoptotic volume decrease in proximal kidney cells

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Role of glutathione transport processes in kidney function

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Protection of NRK-52E cells, a rat renal proximal tubular cell line, from chemical-induced apoptosis by overexpression of a mitochondrial glutathione transporter

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Glutathione depletion and increased apoptosis rate in human cystinotic proximal tubular cells

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Thiazolidinone CFTR inhibitor identified by high-throughput screening blocks cholera toxin-induced intestinal fluid secretion.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Normotonic cell shrinkage because of disordered volume regulation is an early prerequisite to apoptosis

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

DNA fragmentation is a feature of cystic fibrosis epithelial cells: a disease with inappropriate apoptosis?

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Discovery of glycine hydrazide pore-occluding CFTR inhibitors: mechanism, structure-activity analysis, and in vivo efficacy

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Receptor-mediated control of regulatory volume decrease (RVD) and apoptotic volume decrease (AVD).

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Volume-sensitive chloride channels involved in apoptotic volume decrease and cell death.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Scaffold mediated regulation of MAPK signaling and cytoskeletal dynamics: a perspective.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Glutathione: toxicological implications

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

A role of reactive oxygen species in apoptotic activation of volume-sensitive Cl(-) channel

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Molecular dissection of the butyrate action revealed the involvement of mitogen-activated protein kinase in cystic fibrosis transmembrane conductance regulator biogenesis.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Mitochondria as regulators of apoptosis: doubt no more.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Chloride channel inhibition prevents ROS-dependent apoptosis induced by ischemia-reperfusion in mouse cardiomyocytes.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

Mitochondrial control of nuclear apoptosis.

1 reference

https://api.crossref.org/works/10.1152%2FAJPRENAL.00286.2009

21 January 2018

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